Evidence map›Paper›PMID 29592785›Full record

ArticleMolecular pain

Increased expression of Ca

Xue-Jing Kang, Ye-Nan Chi, Wen Chen, Feng-Yu Liu, Shuang Cui, Fei-Fei Liao, Jie Cai, You Wan

Abstract read
In one paragraph

Article in Molecular pain. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Role of HDAC5 Epigenetics in Chronic Craniofacial Neuropathic Pain.International journal of molecular sciences · 2024
    Review
  4. Article
  5. Review
  6. Article
  7. Voltage-dependent CaPflugers Archiv : European journal of physiology · 2022
    Review
  8. Article
  9. Article
  10. Review
  11. Article
  12. Targeting T-type/CaV3.2 channels for chronic pain.Translational research : the journal of laboratory and clinical medicine · 2021
    Review
  13. Review
  14. Review
  15. Article
  16. Article
  17. Contribution of T-Type Calcium Channels to Spinal Cord Injury-Induced Hyperexcitability of Nociceptors.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2020
    Article
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Xue-Jing Kang1 Neuroscience Research Institute, Peking University, Beijing, China.
Ye-Nan Chi1 Neuroscience Research Institute, Peking University, Beijing, China.
Wen Chen1 Neuroscience Research Institute, Peking University, Beijing, China.
Feng-Yu Liu1 Neuroscience Research Institute, Peking University, Beijing, China.
Shuang Cui1 Neuroscience Research Institute, Peking University, Beijing, China.
Fei-Fei Liao1 Neuroscience Research Institute, Peking University, Beijing, China.
Jie Cai1 Neuroscience Research Institute, Peking University, Beijing, China.
You Wan1 Neuroscience Research Institute, Peking University, Beijing, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ion channels are very important in the peripheral sensitization in neuropathic pain. Our present study aims to investigate the possible contribution of Ca

Indexed as

Action PotentialsAnimalsCalcium Channels, T-TypeCell MembraneCell SizeGanglia, SpinalGene SilencingHyperalgesiaMaleNerve TissueNeuralgiaNeuronsNociceptionRats, Sprague-DawleyRNA, AntisenseUp-RegulationCacna1h protein, ratCalcium Channels, T-TypeRNA, AntisenseCaV3.2 T-type calcium channelsdorsal root ganglionneuropathic painspared nerve injury

Identifiers

PMID29592785
PMCPMC5888807

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.