ArticleMatrix biology : journal of the International Society for Matrix Biology2018
Contribution of metabolic disease to bone fragility in MAGP1-deficient mice.
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Article in Matrix biology : journal of the International Society for Matrix Biology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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0cells of the map it votes in
11citing papers in PubMed
1.3field-weighted citation impact, top 18% of its field
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Who cites it
11 citing papers in PubMed, 13 citations in OpenAlex.
- Article
- OIP5-AS1/miR-455-3p/microfibril-associated protein 2 axis exacerbates the progression of thyroid carcinoma.Translational cancer research · 2024Article
- The use of MFAP2 for diagnosis, prognosis and immunotherapy of triple-negative breast cancer.American journal of translational research · 2024Article
- MFAP2 promotes the progression of oral squamous cell carcinoma by activating the Wnt/β-catenin signaling pathway through autophagy.Acta biochimica et biophysica Sinica · 2023Article
- MFAP2 aggravates tumor progression through activating FOXM1/β-catenin-mediated glycolysis in ovarian cancer.The Kaohsiung journal of medical sciences · 2022Article
- Pan-Cancer Analysis of Microfibrillar-Associated Protein 2 (MFAP2) Based on Bioinformatics and qPCR Verification.Journal of oncology · 2022Article
- Long non-coding RNA-KCNQ1OT1 mediates miR-423-5p/microfibril-associated protein 2 axis in colon adenocarcinoma.Histology and histopathology · 2021Article
- Decreased Levels of Microfibril-Associated Glycoprotein (MAGP)-1 in Patients with Colon Cancer and Obesity Are Associated with Changes in Extracellular Matrix Remodelling.International journal of molecular sciences · 2021Article
- A landscape of circulating long non-coding RNA (lncRNA) expression profile and the predictive value of candidate lncRNAs for disease risk of knee osteoarthritis.Journal of clinical laboratory analysis · 2020Article
- Microfibril-Associated Protein 2 (MFAP2) Potentiates Invasion and Migration of Melanoma by EMT and Wnt/β-Catenin Pathway.Medical science monitor : international medical journal of experimental and clinical research · 2020Article
- MFAP2 is overexpressed in gastric cancer and promotes motility via the MFAP2/integrin α5β1/FAK/ERK pathway.Oncogenesis · 2020Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
12 authors at 2 institutions in 2 countries.
S E TurecamoMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: sturecamo@wustl.edu.
T A WaljiMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA; Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA. Electronic address: Tezin.Walji@UTSouthwestern.edu.
T J BroekelmannCell Biology and Physiology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: tbroekel@wustl.edu.
J W WilliamsPathology and Immunology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: jesse.williams@wustl.edu.
S IvanovINSERM U1065, Mediterranean Center of Molecular Medicine, University of Nice Sophia-Antipolis, Faculty of Medicine, Nice, France. Electronic address: stoyan.ivanov@unice.fr.
N K WeeMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA; Department of Reconstructive Sciences, University of Connecticut Health Center, Farmington, CT 06030, USA. Electronic address: wee@uchc.edu.
J D ProcknowMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: jdprocknow@gmail.com.
M R McManusMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: mcmanusm@wustl.edu.
G J RandolphPathology and Immunology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: gjrandolph@wustl.edu.
E L SchellerMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA; Cell Biology and Physiology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: scheller@wustl.edu.
R P MechamCell Biology and Physiology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: bmecham@wustl.edu.
C S CraftMedicine, Division of Bone and Mineral Diseases, Washington University School of Medicine, Saint Louis, MO 63110, USA; Cell Biology and Physiology, Washington University School of Medicine, Saint Louis, MO 63110, USA. Electronic address: clarissa.craft@wustl.edu.
Washington University in St. Louis · USInserm · FR
Funding
KOMP2-Phase3 Production and Phenotyping by the DTCC Consortium TCP Transfer to UC DavisUM1OD023221 · OD · UNIVERSITY OF CALIFORNIA AT DAVIS · PI KC KENT LLOYD · 2016 to 2026
$45.7MWashington University Nutrition Obesity Research CenterP30DK056341 · NIDDK · WASHINGTON UNIVERSITY · PI Brian N Finck · 1999 to 2026
$30.2MWU P&FP30DK020579 · NIDDK · WASHINGTON UNIVERSITY · PI Jennifer Lynn Powers Carson · 2013 to 2026
$27.1MThe Role of the Osteoblast Secretome in Bone FormationP30AR057235 · NIAMS · WASHINGTON UNIVERSITY · PI JOHNSON, AARON N · 2009 to 2018
$5.7MASSEMBLY OF LUNG ELASTINR37HL053325 · NHLBI · WASHINGTON UNIVERSITY · PI MECHAM, ROBERT P. · 2000 to 2009
$4.1MASSEMBLY OF LUNG ELASTINR01HL053325 · NHLBI · WASHINGTON UNIVERSITY · PI MECHAM, ROBERT P. · 1995 to 2019
$3.6MVESSEL STIFFENING, HYPERTENSION, AND VASCULAR EXTRACELLULAR MATRIXR01HL105314 · NHLBI · WASHINGTON UNIVERSITY · PI MECHAM, ROBERT P., WAGENSEIL, JESSICA · 2010 to 2018
$3.2MPathways that regulate monocyte/dendritic cell migrationR01AI061741 · NIAID · WASHINGTON UNIVERSITY · PI RANDOLPH, GWENDALYN J · 2007 to 2011
$2.0MNeural regulation of skeletal biology and periodontal disease progression in typeR00DE024178 · NIDCR · WASHINGTON UNIVERSITY · PI SCHELLER, ERICA LYNN · 2016 to 2018
$735kNeural regulation of skeletal biology and periodontal disease progression in typeK99DE024178 · NIDCR · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI SCHELLER, ERICA LYNN · 2014 to 2015
$203kNHLBI NIH HHS R01 HL053325NHLBI NIH HHS R01 HL105314NHLBI NIH HHS R37 HL053325NIAID NIH HHS R01 AI061741NIAMS NIH HHS P30 AR057235NIDCR NIH HHS K99 DE024178NIDCR NIH HHS R00 DE024178NIDDK NIH HHS P30 DK020579NIDDK NIH HHS P30 DK056341NIH HHS UM1 OD023221
Abstract
Microfibril-associated glycoprotein-1 (MAGP1) is an extracellular matrix protein that interacts with fibrillin and is involved in regulating the bioavailability of signaling molecules such as TGFβ. Mice with germline MAGP1 deficiency (Mfap2
Indexed as
AdipocytesAnimalsBone and BonesBone MarrowContractile ProteinsDisease Models, AnimalExtracellular Matrix ProteinsGerm-Line MutationHomeodomain ProteinsMetabolic DiseasesMiceRNA Splicing FactorsSignal TransductionContractile ProteinsExtracellular Matrix ProteinsHomeodomain Proteinsmicrofibrillar proteinPrrx1 protein, mouseRNA Splicing FactorsBoneBone fractureDiabetesFibrillinMAGP1Marrow adipose tissueMicrofibrilsObesitySkeletal fragility
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.