Evidence map›Paper›PMID 29476190›Full record

ArticleJournal of human genetics2018

Pattern of TSC1 and TSC2 germline mutations in Russian patients with tuberous sclerosis.

Evgeny N Suspitsin, Grigoriy A Yanus, Marina Yu Dorofeeva, Tatiana A Ledashcheva, Nataliya V Nikitina, Galina V Buyanova, Elena V Saifullina, Anna P Sokolenko, Evgeny N Imyanitov

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Article in Journal of human genetics, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.8field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 13 citations in OpenAlex.

  1. Review
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  4. Observational
  5. Hereditary Renal Cancer Syndromes.Medical sciences (Basel, Switzerland) · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 6 institutions in 1 country.

Evgeny N SuspitsinSt. Petersburg Pediatric Medical University, St. Petersburg, Russia. evgeny.suspitsin@gmail.com.ORCID http://orcid.org/0000-0001-9764-2090
Grigoriy A YanusSt. Petersburg Pediatric Medical University, St. Petersburg, Russia.
Marina Yu DorofeevaCenter of Epileptology, N.N. Pirogov National Research Medical University, Moscow, Russia.
Tatiana A LedashchevaCity Center of Medical Genetics, St. Petersburg, Russia.
Nataliya V NikitinaClinical center "Mother and Child Health Protection", Ekaterinburg, Russia.
Galina V BuyanovaPediatric Regional Hospital, Chelyabinsk, Russia.
Elena V SaifullinaPerinatal Center of Republic of Baschkortostan, Ufa, Russia.
Anna P SokolenkoSt. Petersburg Pediatric Medical University, St. Petersburg, Russia.
Evgeny N ImyanitovSt. Petersburg Pediatric Medical University, St. Petersburg, Russia.
Institute of Oncology NN Petrov · RUAcademy of Sciences of the Republic of Bashkortostan · RUEkaterinburg Research Institute of Viral Infections · RUPirogov Russian National Research Medical University · RURegional Clinical Hospital No 4 · RUSt Petersburg University · RU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tuberous sclerosis (TS) is a rare autosomal-dominant genetic disease. TS is manifested by the development of multiple hamartomas, which affect brain, kidneys, retina, skin and other organs. This study aimed to reveal specific features of molecular epidemiology of TS in Russia. Blood DNA samples from 61 patients with definite (n = 53) or probable (n = 8) clinical diagnosis of TS were tested for mutations in TSC1 and TSC2 genes using Sanger sequencing and MLPA analysis. Five TSC1/2 mutation-negative patients were further analyzed by exome sequencing. TSC1/2 mutations were detected in 53/61 patients (87%): 39 (74%) carried mutations in the TSC2 and 14 (26%) in the TSC1. Large rearrangements (exon deletions/duplications) affected exclusively TSC2, accounting for 15% of lesions of this gene. 6/8 (75%) patients with incomplete clinical manifestation of TS carried TSC1/2 gene lesion. Overall, 96% of detected germline TSC1/2 mutations occurred de novo. Patients with no mutation identified (NMI) differed from TSC1/2 mutation carriers, being lacking cortical tubers and subependymal nodules but having higher frequencies of renal angiomyolipomas, rhabdomyomas, and lymphangioleiomyomatosis. Exome sequencing failed to identify overt disease-causing mutation candidates among NMI patients. Russian patients with TS have increased frequency of TSC2 large gene rearrangements and TSC1/2 mutations occurring de novo as compared to other studies. Patients with suspected TS diagnosis but NMI status may represent a distinct disease entity.

Indexed as

Germ-Line MutationAdolescentAdultChildChild, PreschoolDNA Mutational AnalysisExome SequencingFemaleGenetic Association StudiesHumansInfantMalePhenotypeRussiaTuberous SclerosisTuberous Sclerosis Complex 1 ProteinTSC1 protein, humanTSC2 protein, humanTuberous Sclerosis Complex 1 ProteinTuberous Sclerosis Complex 2 ProteinTumor Suppressor Proteins

Identifiers

PMID29476190
OpenAlexW2790677015

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.