ArticlePlant signaling & behavior2018
MLK1 and MLK2 integrate gibberellins and circadian clock signaling to modulate plant growth.
Article in Plant signaling & behavior, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
5 citing papers in PubMed, 6 citations in OpenAlex.
- Article
- The Expression and Function of Circadian Rhythm Genes in Hepatocellular Carcinoma.Oxidative medicine and cellular longevity · 2021Review
- Phosphorylation of Histone H2A at Serine 95 Is Essential for Flowering Time and Development inFrontiers in plant science · 2021Article
- Review
- SIP1 participates in regulation of flowering time in rice by recruiting OsTrx1 to Ehd1.The New phytologist · 2018Article
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The covalent histone modifications were associated with plant development. However, the function of histone modification factors involved in gibberellins (GAs) signaling pathway remains unclear. In recent study, we reported that histone modification factors MUT9p-LIKE KINASE1 (MLK1) and MLK2 coordinate GA and circadian clock signaling in hypocotyl elongation. MLK1 and MLK2 interact with the DELLA protein REPRESSOR OF ga1-3 (RGA), and antagonize the function of RGA to interact with CIRCADIAN CLOCK ASSOCIATED1 (CCA1), resulting in promoting hypocotyl elongation. In this addendum to the report, we presented and discussed the results related to the function of MLK1 and MLK2 in GA pathway. MLK1 and MLK2 interact with RGA, which is independent on 17-amino acid DELLA, TVHYNP, or Poly S/T/V motif, suggesting that MLK1 and MLK2 might have novel functions beyond the protein degradation.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.