Evidence map›Paper›PMID 29392410›Full record

ArticleCurrent genetics2018

STEEx, a boundary between the world of quiescence and the vegetative cycle.

Laetitia Maestroni, Vincent Géli, Stéphane Coulon

Abstract read
PubMed Publisher
In one paragraph

Article in Current genetics, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.7field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Review
  3. Quiescence, an individual journey.Current genetics · 2019
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Laetitia MaestroniCancer Research Institute of Marseille (CRCM), CNRS, INSERM, Aix Marseille Univ, Institut Paoli-Calmettes, Equipe labélisée Ligue contre le cancer, 13273, Marseille, France.
Vincent GéliCancer Research Institute of Marseille (CRCM), CNRS, INSERM, Aix Marseille Univ, Institut Paoli-Calmettes, Equipe labélisée Ligue contre le cancer, 13273, Marseille, France. vincent.geli@inserm.fr.
Stéphane CoulonCancer Research Institute of Marseille (CRCM), CNRS, INSERM, Aix Marseille Univ, Institut Paoli-Calmettes, Equipe labélisée Ligue contre le cancer, 13273, Marseille, France. stephane.coulon@inserm.fr.ORCID http://orcid.org/0000-0001-8090-914X
Centre National de la Recherche Scientifique · FR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Telomere maintenance mechanism is poorly studied in quiescence, a reversible non-proliferative state. We previously described in fission yeast a new mode of repair of telomeres named STEEx, that specifically operates in post-mitotic cells harboring eroded telomeres. This mechanism, promoted by transcription-induced telomeric recombination, prevents cells to exit properly from quiescence, suggesting that STEEx act as an anti-proliferative barrier. Here, we further showed that STEEx are genetically controlled by the Tel1

Indexed as

Cell CycleCell Cycle ProteinsCell DivisionMutationPhosphorylationProtein KinasesSchizosaccharomycesTelomeraseTelomereTelomere HomeostasisCell Cycle ProteinsProtein KinasesTelomeraseDNA damage responseFission yeastQuiescenceSenescenceTelomere

Identifiers

PMID29392410
OpenAlexW2790307954

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.