Evidence map›Paper›PMID 29335582›Full record

ArticleScientific reports2018

Differential roles of Na

Mingyu Ye, Jun Yang, Cuiping Tian, Qiyu Zhu, Luping Yin, Shan Jiang, Mingpo Yang, Yousheng Shu

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
2.6field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 60 citations in OpenAlex.

  1. Article
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  6. Review
  7. Article
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  13. Ataxia and Diplopia: A NewNeurology. Genetics · 2023
    Article
  14. Article
  15. Structure of human NaNature communications · 2023
    Article
  16. Review
  17. Article
  18. Article
  19. Hippocampal astrocytic neogenin regulating glutamate uptake, a critical pathway for preventing epileptic response.Proceedings of the National Academy of Sciences of the United States of America · 2021
    Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 1 country.

Mingyu YeInstitute of Neuroscience, State Key Laboratory of Neuroscience, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China. my2406@cumc.columbia.edu.ORCID http://orcid.org/0000-0001-8560-4345
Jun YangState Key Laboratory of Cognitive Neuroscience and Learning, School of Brain and Cognitive Sciences, the Collaborative Innovation Center for Brain Science, Beijing Normal University, Beijing, China.
Cuiping TianiHuman Institute, ShanghaiTech University, Shanghai, China.
Qiyu ZhuBrain Institute, College of Pharmaceutical Sciences, Capital Medical University, Beijing, China.
Luping YinState Key Laboratory of Cognitive Neuroscience and Learning, School of Brain and Cognitive Sciences, the Collaborative Innovation Center for Brain Science, Beijing Normal University, Beijing, China.ORCID http://orcid.org/0000-0002-8553-6693
Shan JiangInstitute of Neuroscience, State Key Laboratory of Neuroscience, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
Mingpo YangInstitute of Neuroscience, State Key Laboratory of Neuroscience, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
Yousheng ShuState Key Laboratory of Cognitive Neuroscience and Learning, School of Brain and Cognitive Sciences, the Collaborative Innovation Center for Brain Science, Beijing Normal University, Beijing, China. yousheng@bnu.edu.cn.ORCID http://orcid.org/0000-0002-2834-2876
Beijing Normal University · CNChinese Academy of Sciences · CNCapital Medical University · CNShanghaiTech University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Dysregulation of voltage-gated sodium channels (VGSCs) is associated with multiple clinical disorders, including febrile seizures (FS). The contribution of different sodium channel subtypes to environmentally triggered seizures is not well understood. Here we demonstrate that somatic and axonal sodium channels primarily mediated through Na

Indexed as

Action PotentialsAnimalsDisease Models, AnimalMice, KnockoutNAV1.2 Voltage-Gated Sodium ChannelNAV1.6 Voltage-Gated Sodium ChannelNeuronsSeizures, FebrileTemperatureNAV1.2 Voltage-Gated Sodium ChannelNAV1.6 Voltage-Gated Sodium ChannelScn8a protein, mouse

Identifiers

PMID29335582
PMCPMC5768682
OpenAlexW2783055853

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.