Evidence map›Paper›PMID 29332300›Full record

ArticleMetabolic brain disease2018

Long term exposure to cell phone frequencies (900 and 1800 MHz) induces apoptosis, mitochondrial oxidative stress and TRPV1 channel activation in the hippocampus and dorsal root ganglion of rats.

Kemal Ertilav, Fuat Uslusoy, Serdar Ataizi, Mustafa Nazıroğlu

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Article in Metabolic brain disease, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
6.2field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 43 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Kemal ErtilavDepartmant of Neurosurgery, Faculty of Medicine, Suleyman Demirel University, Isparta, Turkey.
Fuat UslusoyDepartment of Plastic Reconstructive and Aesthetic Surgery, Faculty of Medicine, Suleyman Demirel University, Isparta, Turkey.
Serdar AtaiziDepartmant of Neurosurgery, Yunusemre General State Hospital, Eskişehir, Turkey.
Mustafa NazıroğluNeuroscience Research Center, Suleyman Demirel University, TR-32260, Isparta, Turkey. mustafanaziroglu@sdu.edu.tr.
Süleyman Demirel University · TREskişehir City Hospital · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mobile phone providers use electromagnetic radiation (EMR) with frequencies ranging from 900 to 1800 MHz. The increasing use of mobile phones has been accompanied by several potentially pathological consequences, such as neurological diseases related to hippocampal (HIPPON) and dorsal root ganglion neuron (DRGN). The TRPV1 channel is activated different stimuli, including CapN, high temperature and oxidative stress. We investigated the contribution TRPV1 to mitochondrial oxidative stress and apoptosis in HIPPON and DRGN following long term exposure to 900 and 1800 MHz in a rat model. Twenty-four adult rats were equally divided into the following groups: (1) control, (2) 900 MHz, and (3) 1800 MHz exposure. Each experimental group was exposed to EMR for 60 min/ 5 days of the week during the one year. The 900 and 1800 MHz EMR exposure induced increases in TRPV1 currents, intracellular free calcium influx (Ca

Indexed as

Cell PhoneAnimalsApoptosisCalciumFemaleGanglia, SpinalHippocampusMitochondriaNeuronsOxidative StressRats, WistarReactive Oxygen SpeciesTRPV Cation ChannelsCalciumReactive Oxygen SpeciesTrpv1 protein, ratTRPV Cation ChannelsApoptosisDorsal root ganglionHippocampusMobile phone frequencyOxidative stressTRPV1 cation channel

Identifiers

PMID29332300
OpenAlexW2783521028

What OpenQuestion holds

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Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.