Evidence map›Paper›PMID 29322081›Full record

ArticleData in brief2018

Data of expression status of miR- 29a and its putative target mitochondrial apoptosis regulatory gene DRP1 upon miR-15a and miR-214 inhibition.

Muhammad Ishtiaq Jan, Riaz Anwar Khan, Abdul Malik, Tahir Ali, Muhammad Bilal, Long Bo, Abdul Sajid, Naseeb Urehman, Nayyar Waseem, Javed Nawab and 9 more

Open access · goldAbstract read
In one paragraph

Article in Data in brief, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.4field-weighted citation impact, top 42% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 9 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

19 authors at 4 institutions in 2 countries.

Muhammad Ishtiaq JanSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Riaz Anwar KhanDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Abdul MalikDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Tahir AliSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Muhammad BilalLivestock and Dairy Development, Veterinary Research Institute, Peshawar, Pakistan.
Long BoPeking Union medical College, Beijing, China.
Abdul SajidLivestock and Dairy Development, Veterinary Research Institute, Peshawar, Pakistan.
Naseeb UrehmanDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Nayyar WaseemDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Javed NawabDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Murad AliDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Abdul MajeedDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Hamid AhmadDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Sohail AslamDepartment of Cardiovascular Surgery, Lady Reading Hospital Peshawar, Pakistan.
Sadia HameraLUMS, Department of Biology, SBASSE, Lahore, Pakistan.
Aneesa SultanSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Mariam AneesaSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Qamar JavedSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Iram MurtazaSignal Transduction Lab, Department of Bio-Chemistry, Faculty of Biological Sciences, Quaid-i-Azam University Islamabad, Islamabad, Pakistan.
Lady Reading Hospital · PKQuaid-i-Azam University · PKLahore University of Management Sciences · PKPeking Union Medical College Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Data is about the mitochondrial apoptosis regulatory framework genes PUMA, DRP1 (apoptotic), and ARC (anti-apoptotic) analysis after the employment of their controlling miRNAs inhibitors. The data represents putative conserved targeting of seed regions of miR-15a, miR-29a, and miR-214 with respective target genes PUMA, DRP1, and ARC. Data is of cross interference in expression levels of one miRNA family, miR-29a and its putative target DRP1 upon the inhibitory treatment of other miRNAs 15a and 214.

Indexed as

ApoptosisDRP1miR-15amiRNAs inhibition

Identifiers

PMID29322081
PMCPMC5752092
OpenAlexW2776303159

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.