Evidence map›Paper›PMID 29315555›Full record

ReviewImmunology2018

Autoimmunity and autoimmune co-morbidities in psoriasis.

Kazuhisa Furue, Takamichi Ito, Gaku Tsuji, Takafumi Kadono, Takeshi Nakahara, Masutaka Furue

Open access · bronzeAbstract readReview
In one paragraph

Review in Immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 33 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
33citing papers in PubMed, 1 pooled it
3.2field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

33 citing papers in PubMed, 1 synthesis or guideline pooled it, 71 citations in OpenAlex.

  1. Pooled it
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  18. An Analysis ofJournal of clinical medicine · 2021
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Kazuhisa FurueDepartment of Dermatology, Kyushu University, Fukuoka, Japan.
Takamichi ItoDepartment of Dermatology, Kyushu University, Fukuoka, Japan.
Gaku TsujiDepartment of Dermatology, Kyushu University, Fukuoka, Japan.
Takafumi KadonoDepartment of Dermatology, St Marianna University School of Medicine, Kawasaki, Japan.
Takeshi NakaharaDepartment of Dermatology, Kyushu University, Fukuoka, Japan.
Masutaka FurueDepartment of Dermatology, Kyushu University, Fukuoka, Japan.ORCID 0000-0002-2967-1073
Kyushu University · JPSt. Marianna University School of Medicine · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis is characterized by widespread scaly erythematous plaques that cause significant physical and psychological burdens for the affected individuals. Accelerated inflammation driven by the tumour necrosis factor-α/interleukin-23/interleukin-17 axis is now known to be the major mechanism in the development of psoriasis. In addition, psoriasis has an autoimmune nature that manifests as autoreactive T cells and is co-morbid with other autoimmune diseases, such as autoimmune bullous diseases, vitiligo, alopecia and thyroiditis. In this article, we review the recent topics on autoimmunity and autoimmune co-morbidities in psoriasis.

Indexed as

AutoimmunityAnimalsAutoimmune DiseasesComorbidityHumansInterleukin-17Interleukin-23Lymphocyte ActivationPsoriasisSignal TransductionT-LymphocytesTumor Necrosis Factor-alphaInterleukin-17Interleukin-23Tumor Necrosis Factor-alphaalopeciaautoimmunitybullous pemphigoidpsoriasisthyroiditisvitiligo

Identifiers

PMID29315555
PMCPMC5904708
OpenAlexW2783573585

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.