Evidence map›Paper›PMID 29308444›Full record

ArticleJournal of the Endocrine Society2017

High-Fat, High-Sugar Diet Disrupts the Preovulatory Hormone Surge and Induces Cystic Ovaries in Cycling Female Rats.

Katrina M Volk, Veronika V Pogrebna, Jackson A Roberts, Jennifer E Zachry, Sarah N Blythe, Natalia Toporikova

Registry-linked trialOpen access · goldAbstract read
In one paragraph

Article in Journal of the Endocrine Society, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT04801173 (Effect of Very Low Calorie Ketogenic Diet on Obese and Insulin-resistant Women With Polycystic Ovary Syndrome), which is not on this map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
3.9field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT04801173 nacompletednot on this mapstarted 2021, after this paper: background citation

Effect of Very Low Calorie Ketogenic Diet on Obese and Insulin-resistant Women With Polycystic Ovary Syndrome: a Controlled Randomized Trial

TypeinterventionalSponsorIRCCS Azienda Ospedaliero-Universitaria di BolognaRan2021 to 2021Enrolled30ConditionsPCOS, Obesity, Insulin ResistanceArmsVery low calorie ketogenic diet, Low calorie standard diet
3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 39 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Katrina M VolkNeuroscience Program, Washington and Lee University, Lexington, Virginia 24450.
Veronika V PogrebnaDepartment of Biology, Washington and Lee University, Lexington, Virginia 24450.
Jackson A RobertsNeuroscience Program, Washington and Lee University, Lexington, Virginia 24450.
Jennifer E ZachryNeuroscience Program, Washington and Lee University, Lexington, Virginia 24450.
Sarah N BlytheNeuroscience Program, Washington and Lee University, Lexington, Virginia 24450.
Natalia ToporikovaNeuroscience Program, Washington and Lee University, Lexington, Virginia 24450.
Washington and Lee University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diet-induced obesity has been associated with various metabolic and reproductive disorders, including polycystic ovary syndrome. However, the mechanisms by which obesity influences the reproductive system are still not fully known. Studies have suggested that impairments in hormone signaling are associated with the development of symptoms such as acyclicity and ovarian cysts. However, these studies have often failed to address how these hormonal changes arise and how they might contribute to the progression of reproductive diseases. In the present study, we used a high-fat, high-sugar (HFHS) diet to induce obesity in a female rodent model to determine the changes in critical reproductive hormones that might contribute to the development of irregular estrous cycling and reproductive cycle termination. The HFHS animals exhibited impaired estradiol, progesterone (P4), and luteinizing hormone (LH) surges before ovulation. The HFHS diet also resulted in altered basal levels of testosterone (T) and LH. Furthermore, alterations in the basal P4/T ratio correlated strongly with ovarian cyst formation in HFHS rats. Thus, this model provides a method to assess the underlying etiology of obesity-related reproductive dysfunction and to examine an acyclic reproductive phenotype as it develops.

Indexed as

decreased luteinizing hormonehormone surgeincreased estradiolobesityovarian cyststestosterone

Identifiers

PMID29308444
PMCPMC5740526
OpenAlexW2766860077

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.