Evidence map›Paper›PMID 29260245›Full record

ReviewDer Nervenarzt2018

[Cerebral amyloid angiopathy associated with inflammation].

J Schaumberg, M Trauscheid, B Eckert, D Petersen, W Schulz-Schaeffer, J Röther, W Heide

Abstract readCase ReportsReview
PubMed Publisher
In one paragraph

Review in Der Nervenarzt, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.2field-weighted citation impact, top 41% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
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  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 4 institutions in 1 country.

J SchaumbergNeurologie, Asklepios Klinik Altona, Paul-Ehrlich-Straße 1, 22763, Hamburg, Deutschland. j.schaumberg@web.de.
M TrauscheidNeurologie, Allgemeines Krankenhaus Celle, Celle, Deutschland.
B EckertNeuroradiologie, Asklepios Klinik Altona, Hamburg, Deutschland.
D PetersenInstitut für Neuroradiologie, Universität zu Lübeck, Lübeck, Deutschland.
W Schulz-SchaefferInstitut für Neuropathologie, Universitätsklinikum des Saarlandes, Homburg, Deutschland.
J RötherNeurologie, Asklepios Klinik Altona, Paul-Ehrlich-Straße 1, 22763, Hamburg, Deutschland.
W HeideNeurologie, Allgemeines Krankenhaus Celle, Celle, Deutschland.
Asklepios Klinik Altona · DEAllgemeines Krankenhaus Celle · DEUniversitätsklinikum des Saarlandes · DEUniversity of Lübeck · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cerebral amyloid angiopathy (CAA) associated with inflammation is a rare form of a potentially reversible encephalopathy in a subgroup of patients with CAA. The cerebral amyloid deposition can in isolated cases induce an inflammation predominantly of the cerebral blood vessels and a multifocal edema of the cerebral white matter. The courses can occur as monophasic, relapsing remitting and primarily progressive forms. We present seven cases with different courses of the disease and give an overview of the pathophysiology, clinical aspects and treatment of the disease with reference to the current literature. The cases presented show a very different and often difficult differential diagnostic clinical picture and all showed a significant improvement under steroid medication without signs of recurrence of the disease during the course. The recognition and early consistent treatment of inflammatory forms of CAA with and without direct inflammatory involvement of vessels can be decisive for successful treatment.

Indexed as

Cerebral Amyloid AngiopathyBrainHumansInflammationWhite MatterAmyloid-beta related angiitis (ABRA)Cerebral amyloid angiopathy (CAA)InflammationPrimary angiitis of the central nervous systemSuperficial siderosis

Identifiers

PMID29260245
OpenAlexW2777089426

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.