Evidence map›Paper›PMID 29255176›Full record

ArticleEuropean journal of human genetics : EJHG2017

Repeat genetic testing with targeted capture sequencing in primary arrhythmia syndrome and cardiomyopathy.

Tomas Robyns, Cuno Kuiperi, Jeroen Breckpot, Koenraad Devriendt, Erika Souche, Johan Van Cleemput, Rik Willems, Dieter Nuyens, Gert Matthijs, Anniek Corveleyn

Open access · bronzeAbstract read
In one paragraph

Article in European journal of human genetics : EJHG, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
2.0field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 15 citations in OpenAlex.

  1. Article
  2. Review
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  4. Article
  5. Inherited arrhythmia syndrome predisposing to sudden cardiac death.The Korean journal of internal medicine · 2021
    Review
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  7. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Tomas RobynsDepartment of Cardiovascular Sciences, University of Leuven, Leuven, Belgium. Tomas.Robyns@uzleuven.be.
Cuno KuiperiCenter for Human Genetics, University of Leuven, Leuven, Belgium.
Jeroen BreckpotCenter for Human Genetics, University of Leuven, Leuven, Belgium.
Koenraad DevriendtCenter for Human Genetics, University of Leuven, Leuven, Belgium.ORCID http://orcid.org/0000-0002-3651-2548
Erika SoucheCenter for Human Genetics, University of Leuven, Leuven, Belgium.
Johan Van CleemputDepartment of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Rik WillemsDepartment of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Dieter NuyensDepartment of Cardiology, Hospital Oost-Limburg (ZOL), Genk, Belgium.
Gert MatthijsCenter for Human Genetics, University of Leuven, Leuven, Belgium.
Anniek CorveleynCenter for Human Genetics, University of Leuven, Leuven, Belgium.
KU Leuven · BEZiekenhuis Oost-Limburg · BE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In inherited primary arrhythmia syndromes (PAS) and cardiomyopathies (CMP), the yield of genetic testing varies between 20 and 75% in different diseases according to studies performed in the pre next-generation sequencing (NGS) era. It is unknown whether retesting historical negative samples with NGS techniques is worthwhile. Therefore, we assessed the value of NGS-based panel testing in previously genotype negative-phenotype positive probands. We selected 107 patients (47 PAS and 60 CMP) with a clear phenotype who remained genotype negative after genetic analysis of the main genes implicated in their specific phenotype. Targeted sequencing of the coding regions of 71 PAS- and CMP-related genes was performed. Variant interpretation and classification was done according to a cardiology-specific scoring algorithm ('Amsterdam criteria') and the ACMG-AMP criteria. Co-segregation analysis was performed when DNA and clinical data of family members were available. Finally, a genetic diagnosis could be established in 21 patients (20%), 5 PAS (11%) and 16 CMP (27%) patients, respectively. The increased detection rate was due to sequencing of novel genes in 52% of the cases and due to technical failures with the historical analysis in 48%. A total of 118 individuals were informed about their carrier state and either reassured or scheduled for proper follow-up. To conclude, genetic retesting in clinically overt PAS and CMP cases, who were genotype negative with older techniques, resulted in an additional genetic diagnosis in up to 20% of the cases. This clearly supports a policy for genetic retesting with NGS-based panels.

Indexed as

Arrhythmias, CardiacCardiomyopathiesGenetic LociGenetic TestingHigh-Throughput Nucleotide SequencingHumansPolymorphism, GeneticSensitivity and SpecificitySequence Analysis, DNASyndrome

Identifiers

PMID29255176
PMCPMC5865127
OpenAlexW2764090718

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.