Evidence map›Paper›PMID 29229093›Full record

ReviewRespiratory medicine2017

Bronchopulmonary dysplasia: A review of pathogenesis and pathophysiology.

Renjithkumar Kalikkot Thekkeveedu, Milenka Cuevas Guaman, Binoy Shivanna

Registry-linked trialOpen access · greenAbstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Respiratory medicine, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT03014453 (Gastroesophageal Reflux Poses a Potential Risk for Late Complications of BPD), which is not on this map. Cited by 237 papers, 13 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
237citing papers in PubMed, 13 pooled it
22.3field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT03014453 completednot on this map

Gastroesophageal Reflux Poses a Potential Risk for Late Complications of BPD: A Prospective Study

TypeobservationalSponsorShengjing HospitalRan2017 to 2018Enrolled187ConditionsGastroesophageal Reflux, Bronchopulmonary Dysplasia
3 · Its place in the literature

Who cites it

237 citing papers in PubMed, 13 syntheses or guidelines pooled it, 418 citations in OpenAlex.

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  17. The role of prophylactic arginine supplementation for prevention of bronchopulmonary dysplasia in extremely preterm infants.Journal of perinatology : official journal of the California Perinatal Association · 2026
    Article
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177 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Renjithkumar Kalikkot ThekkeveeduSection of Neonatology, Department of Pediatrics, University of Mississippi, Jackson, MS, USA.
Milenka Cuevas GuamanSection of Neonatology, Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.
Binoy ShivannaSection of Neonatology, Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA. Electronic address: shivanna@bcm.edu.
Baylor College of Medicine · USUniversity of Mississippi · US

Funding

Modulation of Neonatal Hyperoxic Lung Injury by the Aryl Hydrocarbon ReceptorK08HD073323 · NICHD · BAYLOR COLLEGE OF MEDICINE · PI SHIVANNA, BINOY · 2013 to 2017
$649k
NICHD NIH HHS K08 HD073323
6 · The paper itself

Abstract

Bronchopulmonary dysplasia (BPD) is a chronic lung disease of primarily premature infants that results from an imbalance between lung injury and repair in the developing lung. BPD is the most common respiratory morbidity in preterm infants, which affects nearly 10, 000 neonates each year in the United States. Over the last two decades, the incidence of BPD has largely been unchanged; however, the pathophysiology has changed with the substantial improvement in the respiratory management of extremely low birth weight (ELBW) infants. Here we have attempted to comprehensively review and summarize the current literature on the pathogenesis and pathophysiology of BPD. Our goal is to provide insight to help further progress in preventing and managing severe BPD in the ELBW infants.

Indexed as

Bronchopulmonary DysplasiaChorioamnionitisDuctus Arteriosus, PatentFemaleFetal Growth RetardationGenetic Predisposition to DiseaseGestational AgeHumansHyperoxiaHypertension, Pregnancy-InducedIncidenceInfant, Extremely Low Birth WeightInfant, Extremely PrematureInfant, NewbornInfant, PrematureMaleBronchopulmonary dysplasiaHyperoxiaLung functionPathogenesisPathophysiologyVentilator-induced lung injury

Identifiers

PMID29229093
PMCPMC5729938
OpenAlexW2765990633

What OpenQuestion holds

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Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.