Evidence map›Paper›PMID 29211299›Full record

ArticleImmunology2018

Endoplasmic reticulum stress induced LOX-1

Jiang Nan, Yan-Fang Xing, Bo Hu, Jian-Xin Tang, Hui-Min Dong, Yu-Mei He, Dan-Yun Ruan, Qing-Jian Ye, Jia-Rong Cai, Xiao-Kun Ma and 5 more

Open access · bronzeAbstract read
In one paragraph

Article in Immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 81 papers.

0numbers the graph read from it
0cells of the map it votes in
81citing papers in PubMed
3.4field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

81 citing papers in PubMed, 110 citations in OpenAlex.

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21 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

15 authors at 2 institutions in 1 country.

Jiang NanDepartment of Hepatic Surgery, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Yan-Fang XingDepartment of Nephrology, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Bo HuDepartment of Central Laboratory, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Jian-Xin TangDepartment of Hepatic Surgery, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Hui-Min DongDepartment of Central Laboratory, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Yu-Mei HeInstitute of Human Virology, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, China.
Dan-Yun RuanDepartment of Medical Oncology and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Qing-Jian YeDepartment of Gynaecology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Jia-Rong CaiDepartment of Urology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Xiao-Kun MaDepartment of Medical Oncology and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Jie ChenDepartment of Medical Oncology and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Xiu-Rong CaiDepartment of Medical Oncology and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Ze-Xiao LinDepartment of Medical Oncology and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Xiang-Yuan WuDepartment of Liver Surgery and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Xing LiDepartment of Liver Surgery and Guangdong Key Laboratory of Liver Disease Research, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.ORCID 0000-0001-9145-4769
Sun Yat-sen University · CNThird Affiliated Hospital of Guangzhou Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

A recent study indicated that Lectin-type oxidized LDL receptor-1 (LOX-1) was a distinct surface marker for human polymorphisms myeloid-derived suppressor cells (PMN-MDSC). The present study was aimed to investigate the existence LOX-1 PMN-MDSC in hepatocellular carcinoma (HCC) patients. One hundred and twenty-seven HCC patients, 10 patients with mild active chronic hepatitis B, 10 liver cirrhosis due to hepatitis B, 10 liver dysplastic node with hepatitis B and 50 health control were included. LOX-1

Indexed as

Endoplasmic Reticulum StressArginaseCarcinoma, HepatocellularCase-Control StudiesCell ProliferationCells, CulturedCoculture TechniquesFucosyltransferasesHumansInterferonsLewis X AntigenLiver NeoplasmsLymphocyte ActivationMyeloid-Derived Suppressor CellsReactive Oxygen SpeciesScavenger Receptors, Class EARG1 protein, humanArginaseFucosyltransferasesFUT4 protein, humanInterferonsLewis X AntigenOLR1 protein, humanReactive Oxygen SpeciesScavenger Receptors, Class Eendoplasmic reticulum stresshepatocelluar carcinoma patientslectin-type oxidized LDL receptor-1polymorphonuclear myeloid-derived suppressor cellprognosis

Identifiers

PMID29211299
PMCPMC5904716
OpenAlexW2771587821

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.