Evidence map›Paper›PMID 29208635›Full record

ArticleDisease models & mechanisms2018

The

Aya Yoshimura, Naoki Adachi, Hitomi Matsuno, Masaki Kawamata, Yusuke Yoshioka, Hisae Kikuchi, Haruki Odaka, Tadahiro Numakawa, Hiroshi Kunugi, Takahiro Ochiya and 1 more

Open access · goldAbstract read
In one paragraph

Article in Disease models & mechanisms, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 33 citations in OpenAlex.

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  10. Exomap1 mouse: A transgenic model forExtracellular vesicle · 2023
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Aya YoshimuraDivision of Laboratory Animals Resources, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Naoki AdachiDepartment of Mental Disorder Research, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Hitomi MatsunoDepartment of Mental Disorder Research, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Masaki KawamataDivision of Molecular and Cellular Medicine, National Cancer Center Research Institute (NCC), 1-1 Tsukiji 5-chome, Chuo-ku, Tokyo 104-0045, Japan.
Yusuke YoshiokaDivision of Molecular and Cellular Medicine, National Cancer Center Research Institute (NCC), 1-1 Tsukiji 5-chome, Chuo-ku, Tokyo 104-0045, Japan.
Hisae KikuchiDepartment of Degenerative Neurological Diseases, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Haruki OdakaDepartment of Mental Disorder Research, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Tadahiro NumakawaDepartment of Mental Disorder Research, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Hiroshi KunugiDepartment of Mental Disorder Research, National Institute of Neuroscience, NCNP, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Takahiro OchiyaDivision of Molecular and Cellular Medicine, National Cancer Center Research Institute (NCC), 1-1 Tsukiji 5-chome, Chuo-ku, Tokyo 104-0045, Japan tochiya@ncc.go.jp.ORCID 0000-0002-0776-9918
Yoshitaka TamaiDivision of Laboratory Animals Resources, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
National Center of Neurology and Psychiatry · JPKwansei Gakuin University · JPWaseda University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Extracellular vesicles (EVs) can modulate microenvironments by transferring biomolecules, including RNAs and proteins derived from releasing cells, to target cells. To understand the molecular mechanisms maintaining the neural stem cell (NSC) niche through EVs, a new transgenic (Tg) rat strain that can release human CD63-GFP-expressing EVs from the NSCs was established. Human CD63-GFP expression was controlled under the rat

Indexed as

Promoter Regions, GeneticAnimalsAnimals, NewbornAstrocytesBrainCell DifferentiationCoculture TechniquesEmbryo, MammalianExtracellular VesiclesGreen Fluorescent ProteinsHumansModels, AnimalNeural Stem CellsRats, TransgenicSOXB1 Transcription FactorsSpheroids, CellularGreen Fluorescent ProteinsSOXB1 Transcription FactorsTetraspanin 30CD63Extracellular vesiclesModel animalsNeural stem cells

Identifiers

PMID29208635
PMCPMC5818070
OpenAlexW2772866788

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.