Evidence map›Paper›PMID 29176673›Full record

ArticleScientific reports2017

Phosphoproteomics of cAMP signaling of Bordetella adenylate cyclase toxin in mouse dendritic cells.

Jakub Novák, Ivo Fabrik, Irena Linhartová, Marek Link, Ondřej Černý, Jiří Stulík, Peter Šebo

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.7field-weighted citation impact, top 30% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
  2. mBio · 2025
    Article
  3. Bacterial pore-forming toxins.Microbiology (Reading, England) · 2022
    Article
  4. Review
  5. Review
  6. Article
  7. Review
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 2 countries.

Jakub NovákInstitute of Microbiology of the Czech Academy of Sciences, v.v.i., Prague, Czech Republic.
Ivo FabrikDepartment of Molecular Pathology and Biology, Faculty of Military Health Sciences, University of Defence, Hradec Kralove, Czech Republic.
Irena LinhartováInstitute of Microbiology of the Czech Academy of Sciences, v.v.i., Prague, Czech Republic.
Marek LinkDepartment of Molecular Pathology and Biology, Faculty of Military Health Sciences, University of Defence, Hradec Kralove, Czech Republic.
Ondřej ČernýInstitute of Microbiology of the Czech Academy of Sciences, v.v.i., Prague, Czech Republic.
Jiří StulíkDepartment of Molecular Pathology and Biology, Faculty of Military Health Sciences, University of Defence, Hradec Kralove, Czech Republic.
Peter ŠeboInstitute of Microbiology of the Czech Academy of Sciences, v.v.i., Prague, Czech Republic. sebo@biomed.cas.cz.ORCID 0000-0002-9755-7715
Czech Academy of Sciences · CZUniversity of Defence · CZ

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The adenylate cyclase toxin (CyaA) of the whooping cough agent Bordetella pertussis subverts immune functions of host myeloid cells expressing the α

Indexed as

AnimalsBordetella pertussisCell Adhesion MoleculesCyclic AMPCytoskeletal ProteinsDendritic CellsFemaleHistone DeacetylasesMiceMice, Inbred C57BLMicrofilament ProteinsPhosphoproteinsSignal TransductionTalinTranscription FactorsVasodilator-Stimulated PhosphoproteinCell Adhesion MoleculesCRTC3 protein, mouseCyclic AMPCytoskeletal ProteinsEnah protein, mouseHdac5 protein, mouseHistone DeacetylasesMicrofilament ProteinsPhosphoproteinsTalinTln1 protein, mouseTranscription FactorsVasodilator-Stimulated Phosphoprotein

Identifiers

PMID29176673
PMCPMC5701129
OpenAlexW2769891767

What OpenQuestion holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.