Evidence map›Paper›PMID 29159314›Full record

ArticleBiochemistry and biophysics reports2017

Vasoconstriction triggered by hydrogen sulfide: Evidence for Na

Sergei N Orlov, Svetlana V Gusakova, Liudmila V Smaglii, Svetlana V Koltsova, Svetalana V Sidorenko

Open access · goldAbstract read
In one paragraph

Article in Biochemistry and biophysics reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
0.8field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 18 citations in OpenAlex.

  1. Inhibition of HJournal of vascular research · 2026
    Article
  2. Article
  3. HAdvanced science (Weinheim, Baden-Wurttemberg, Germany) · 2024
    Article
  4. Article
  5. Clinical Potential of Hydrogen Sulfide in Peripheral Arterial Disease.International journal of molecular sciences · 2023
    Review
  6. Review
  7. Review
  8. Review
  9. Hydrogen sulfide and vascular regulation - An update.Journal of advanced research · 2021
    Review
  10. The role of gasotransmitters in neonatal physiology.Nitric oxide : biology and chemistry · 2020
    Review
  11. Hydrogen Sulfide, Adipose Tissue and Diabetes Mellitus.Diabetes, metabolic syndrome and obesity : targets and therapy · 2020
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Sergei N OrlovCentral Research Laboratory, Siberian State Medical University, Moskovskiy trakt 2, Tomsk 634050, Russia.
Svetlana V GusakovaDepartment of Biophysics and Functional Diagnostics, Medical and Biological Faculty, Siberian State Medical University, Moskovskiy trakt 2, Tomsk 634050, Russia.
Liudmila V SmagliiDepartment of Biophysics and Functional Diagnostics, Medical and Biological Faculty, Siberian State Medical University, Moskovskiy trakt 2, Tomsk 634050, Russia.
Svetlana V KoltsovaFaculty of Biology MV Lomonosov Moscow State University, Leninskiye gory 1/12, Moscow 119991, Russia.
Svetalana V SidorenkoFaculty of Biology MV Lomonosov Moscow State University, Leninskiye gory 1/12, Moscow 119991, Russia.
Siberian State Medical University · RULomonosov Moscow State University · RU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectivesThis study examined the dose-dependent actions of hydrogen sulfide donor sodium hydrosulphide (NaHS) on isometric contractions and ion transport in rat aorta smooth muscle cells (SMC).

methodsIsometric contraction was measured in ring aortas segments from male Wistar rats. Activity of Na

resultsNaHS exhibited the bimodal action on contractions triggered by modest depolarization ([K

conclusionsOur results strongly suggest that contractions of rat aortic rings triggered by low doses of NaHS are mediated by activation of Na

Indexed as

Ca2+ influxcGMP, cyclic guanosine monophosphateCO, carbon monoxideContractionCOX, cyclooxygenaseCSE, cystathionine-γ-lyaseEC, endothelial cellsEDHF, endothelium-derived hyperpolarizing factorH2S, hydrogen sulfideHydrogen sulfideKATP, ATP-sensitive potassium channelsKCa, Ca2+-activated potassium channelsNaHS, sodium hydrosulphideNa+,K+,2Cl-cotransportNKCC, Na+,K+,2Cl- cotransportNO, nitric oxidePE, phenylephrinePSS, physiologically-balanced salt solutionRAEC, endothelial cells from rat aortaRASMC, smooth muscle cells from rat aortaRat aortasGC, soluble guanylyl cyclaseSMC, smooth muscle cellsSmooth muscle cellsTEA, tetraethylammonium chlorideVSMC, vascular smooth muscle cellsVSMR, vascular smooth muscles from rat

Identifiers

PMID29159314
PMCPMC5683885
OpenAlexW2767560216

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.