Evidence map›Paper›PMID 29084713›Full record

ArticleBiology open2017

Cohesin mediates Esco2-dependent transcriptional regulation in a zebrafish regenerating fin model of Roberts Syndrome.

Rajeswari Banerji, Robert V Skibbens, M Kathryn Iovine

Open access · goldAbstract read
In one paragraph

Article in Biology open, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
1.2field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 23 citations in OpenAlex.

  1. Protein turnover downstream of the Nipbl/CRL4 axis contributes to abnormal development in zebrafish embryos.Developmental dynamics : an official publication of the American Association of Anatomists · 2026
    Article
  2. Article
  3. Article
  4. Article
  5. Retinoic Acid InfluencesBioelectricity · 2023
    Article
  6. Article
  7. Article
  8. Esco2 and cohesin regulate CRL4 ubiquitin ligaseCell cycle (Georgetown, Tex.) · 2022
    Article
  9. Article
  10. Review
  11. Article
  12. Article
  13. Article
  14. Article
  15. Review
  16. Article
  17. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Rajeswari BanerjiDepartment of Biological Science, Lehigh University, Bethlehem, Pennsylvania 18015, USA.
Robert V SkibbensDepartment of Biological Science, Lehigh University, Bethlehem, Pennsylvania 18015, USA rvs3@lehigh.edu mki3@lehigh.edu.
M Kathryn IovineDepartment of Biological Science, Lehigh University, Bethlehem, Pennsylvania 18015, USA rvs3@lehigh.edu mki3@lehigh.edu.ORCID http://orcid.org/0000-0003-0046-8482
Lehigh University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Robert syndrome (RBS) and Cornelia de Lange syndrome (CdLS) are human developmental disorders characterized by craniofacial deformities, limb malformation and mental retardation. These birth defects are collectively termed cohesinopathies as both arise from mutations in cohesion genes. CdLS arises due to autosomal dominant mutations or haploinsufficiencies in cohesin subunits (

Indexed as

CohesinCornelia de Lange syndromecx43esco2RegenerationRoberts Syndromesmc3TranscriptionZebrafish

Identifiers

PMID29084713
PMCPMC5769645
OpenAlexW2766071271

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.