Evidence map›Paper›PMID 28972021›Full record

ArticleJournal of bacteriology2017

Genetic Dissection of DivIVA Functions in Listeria monocytogenes.

Karan Gautam Kaval, Samuel Hauf, Jeanine Rismondo, Birgitt Hahn, Sven Halbedel

Open access · bronzeAbstract read
In one paragraph

Article in Journal of bacteriology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.3field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 20 citations in OpenAlex.

  1. Article
  2. Article
  3. Septum site placement inMicrobiology (Reading, England) · 2023
    Article
  4. Article
  5. Article
  6. EF1025, a Hypothetical Protein FromFrontiers in microbiology · 2020
    Article
  7. ¡vIVA la DivIVA!Journal of bacteriology · 2019
    Review
  8. Article
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Karan Gautam KavalFG11 Division of Enteropathogenic Bacteria and Legionella, Robert Koch Institute, Wernigerode, Germany.
Samuel HaufFG11 Division of Enteropathogenic Bacteria and Legionella, Robert Koch Institute, Wernigerode, Germany.
Jeanine RismondoFG11 Division of Enteropathogenic Bacteria and Legionella, Robert Koch Institute, Wernigerode, Germany.
Birgitt HahnFG11 Division of Enteropathogenic Bacteria and Legionella, Robert Koch Institute, Wernigerode, Germany.
Sven HalbedelFG11 Division of Enteropathogenic Bacteria and Legionella, Robert Koch Institute, Wernigerode, Germany halbedels@rki.de.
Robert Koch Institute · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

DivIVA is a membrane binding protein that clusters at curved membrane regions, such as the cell poles and the membrane invaginations occurring during cell division. DivIVA proteins recruit many other proteins to these subcellular sites through direct protein-protein interactions. DivIVA-dependent functions are typically associated with cell growth and division, even though species-specific differences in the spectrum of DivIVA functions and their causative interaction partners exist. DivIVA from the Gram-positive human pathogen

Indexed as

AllelesBacterial ProteinsCell Cycle ProteinsCell DivisionCell WallListeria monocytogenesN-Acetylmuramoyl-L-alanine AmidasePeptidoglycanProtein TransportBacterial ProteinsCell Cycle ProteinsDivIVA protein, bacteriaN-Acetylmuramoyl-L-alanine AmidasePeptidoglycanautolysin secretioncell divisiondivision site selectionMin systemrough colony phenotype

Identifiers

PMID28972021
PMCPMC5686608
OpenAlexW2762338663

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.