ArticleJournal of neurosciences in rural practice2017
Unilateral Papilledema in Cerebral Venous Sinus Thrombosis.
Article in Journal of neurosciences in rural practice, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
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Who cites it
7 citing papers in PubMed, 12 citations in OpenAlex.
- Unilateral Papilledema with Bilateral Optic Nerve Sheath Distension: A Case Report.Turkish journal of ophthalmology · 2024Article
- Clinical characteristics of cerebral venous sinus thrombosis patients with new-onset of headache.BMC neurology · 2023Article
- Optic Disc Edema and Elevated Intracranial Pressure (ICP): A Comprehensive Review of Papilledema.Cureus · 2022Review
- Clinical profile, diagnostic challenges, and outcomes in subacute/chronic cerebral sinus venous thrombosis.Indian journal of ophthalmology · 2021Observational
- Unilateral persistent disc oedema due to cerebral sinus venous thrombosis (CSVT): diagnostic and management challenge.BMJ case reports · 2020Article
- The relation of optic nerve sheath diameter (ONSD) and intracranial pressure (ICP) in pediatric neurosurgery practice - Part I: Correlations, age-dependency and cut-off values.Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery · 2020Article
- Papilledema: A Comprehensive Assessment.Journal of neurosciences in rural practiceArticle
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Authors and funding
5 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
In the majority of patients with raised intracranial pressure, the papilledema is bilateral. Unilateral papilledema is rare in conditions causing intracranial hypertension, and it has been described in Foster-Kennedy syndrome and in some cases of idiopathic intracranial hypertension. It has never been reported in cerebral venous thrombosis. We report a young lady presenting with features of subacute onset of headache with seizures, on evaluation she had superior sagittal and bilateral lateral sinus thrombosis. The risk factors found on evaluation were Vitamin B12 deficiency and hyperhomocysteinemia. On optic fundus examination, she had swollen optic disc on the right side with normal fundus on the left side, confirmed with the orbital ultrasound B-scan and optic coherence tomography. Her magnetic resonance imaging showed features of raised intracranial pressure with thrombosis of the superior sagittal and bilateral lateral sinus thrombosis. She was treated with anticoagulation (heparin followed by oral anticoagulants), antiedema measures, and vitamin supplementation for hyperhomocysteinemia. She improved over time and was asymptomatic during follow-up. We discuss the possible mechanisms described in the literature for unilateral papilledema. This report highlights the need for carefully performing bilateral fundus examination so as not to miss the vision or life-threatening causes of a headache.
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