ArticleFrontiers in neuroscience2017
Concurrence of Inappropriate Antidiuretic Hormone Secretion and Cerebral Salt Wasting Syndromes after Traumatic Brain Injury.
Article in Frontiers in neuroscience, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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Who cites it
10 citing papers in PubMed.
- Cerebral salt wasting syndrome in an elderly patient with traumatic brain injury: a case report.Frontiers in medicine · 2026Article
- Cerebral Salt-Wasting Syndrome in Severe Brain Trauma: A Case Report and Review of the Literature.Cureus · 2025Article
- Extracranial Effects of Traumatic Brain Injury: A Narrative Review.Clinics and practice · 2025Review
- Is cerebral salt wasting always present in patients with traumatic brain injury?Surgical neurology international · 2025Article
- Late-Onset Intracranial Hemorrhage Presenting as Refractory Hyponatremia: A Case Report.Cureus · 2023Article
- High Urinary Sodium Concentrations in Severe SIADH: Case Reports of 2 Patients and Literature Review.Frontiers in medicine · 2022Article
- Article
- Gradient washout and secondary nephrogenic diabetes insipidus after brain injury in an infant: a case report.Journal of medical case reports · 2020Article
- Inappropriate Antidiuretic Hormone Secretion and Cerebral Salt-Wasting Syndromes in Neurological Patients.Frontiers in neuroscience · 2019Review
- Mechanism, spectrum, consequences and management of hyponatremia in tuberculous meningitis.Wellcome open research · 2019Review
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Authors and funding
3 authors.
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Abstract
Syndrome of inappropriate antidiuretic hormone (SIADH) and cerebral salt wasting syndrome (CSWS) as the two most common neuroendocrine diseases, have been recognized and understood by many neurologists. Although SIADH and CSWS are the common causes of central hyponatremia after traumatic brain injury (TBI), a few cases are mixed, with the coexistence of the two pathological pathomechanism. However, the mixed type of both SIADH and CSWS has not been clearly reported in any literature. Here, we present the first description of the concurrent syndrome of SIADH and CSWS after TBI in four patients who underwent standard diagnostic procedures, treatment and follow up. Our findings further support that this rare neuroendocrine disease may exist in clinical practice, in which the traditional-conventional treatment shows poor efficacy.
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