ArticleThe American journal of case reports2017
A Case of Hepatorenal Syndrome and Abdominal Compartment Syndrome with High Renal Congestion.
Article in The American journal of case reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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Who cites it
5 citing papers in PubMed, 11 citations in OpenAlex.
- From past to present to future: Terlipressin and hepatorenal syndrome-acute kidney injury.Hepatology (Baltimore, Md.) · 2025Review
- Hepatorenal syndrome: Paving a pathway from a fatal condition to an opportunity to preserve kidney function.World journal of nephrology · 2025Article
- Renal venous congestion following hemorrhagic shock due to traumatic liver injury.CEN case reports · 2021Article
- Renal vein measurement using ultrasonography in patients with cirrhotic ascites and congestive heart failure.Journal of medical ultrasonics (2001) · 2021Observational
- Protective Effects of Methane-Rich Saline on Renal Ischemic-Reperfusion Injury in a Mouse Model.Medical science monitor : international medical journal of experimental and clinical research · 2018Article
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Authors and funding
13 authors at 1 institution in 1 country.
Funding
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Abstract
BACKGROUND Hepatorenal syndrome (HRS) is a reversible renal impairment that occurs in patients with acute liver failure and advanced liver cirrhosis. HRS is due to a renal vasoconstriction that results from extreme vasodilatation. It is therefore a functional disorder, not associated with structural kidney damage. On the other hand, end-stage liver diseases are often complicated by massive ascites. Massive ascites may cause abdominal compartment syndrome (ACS), which includes impairment of renal blood flow, but there are no reports indicating that kidney lesions caused by ACS may pathologically contribute to end-stage liver diseases. CASE REPORT A 40-year-old man with acute liver failure was admitted to our hospital. He was diagnosed with type 1 HRS and showed ACS at the same time. He died 30 days after admission. There were signs of congestion in the kidneys upon dissection and advanced erythroid fullness in the renal tubules. CONCLUSIONS We report an autopsy case with HRS and ACS diagnosed with a clinical and histopathological consideration of liver and kidney. Further clinical studies are needed to improve management of renal failure in patients with acute liver failure and advanced liver cirrhosis.
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