Evidence map›Paper›PMID 28870694›Full record

ArticleThe Journal of investigative dermatology2018

Embryonic Fate Mapping Uncovers the Critical Role of microRNAs in the Development of Epidermal γδ T Cells.

Yi Yao, Queping Liu, Carly Martin, Congcong Yin, Zheng Dong, Qing-Sheng Mi, Li Zhou

Abstract readLetter
PubMed Full text at PMC Publisher
In one paragraph

Article in The Journal of investigative dermatology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Article
  2. Review
  3. Review
  4. Epidermal resident γδ T cell development and function in skin.Cellular and molecular life sciences : CMLS · 2021
    Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Yi YaoCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA.
Queping LiuCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Carly MartinCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA.
Congcong YinCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA.
Zheng DongDepartment of Cellular Biology and Anatomy, University of Augusta and Charlie Norwood VA Medical Center, Augusta, Georgia, USA.
Qing-Sheng MiCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA; Department of Internal Medicine, Henry Ford Health System, Detroit, Michigan, USA. Electronic address: QMI1@hfhs.org.
Li ZhouCenter for Cutaneous Biology and Immunology Research, Department of Dermatology, Henry Ford Health System, Detroit, Michigan, USA; Immunology Research Program, Henry Ford Cancer Institute, Henry Ford Health System, Detroit, Michigan, USA; Department of Internal Medicine, Henry Ford Health System, Detroit, Michigan, USA. Electronic address: lzhou1@hfhs.org.

Funding

microRNAs and NKT cell development and functionR01AI119041 · NIAID · HENRY FORD HEALTH SYSTEM · PI MI, QING-SHENG · 2016 to 2020
$1.8M
Roles of HDAC3 in Epidermal Langerhans Cell Ontogeny and FunctionR01AR069681 · NIAMS · HENRY FORD HEALTH SYSTEM · PI MI, QING-SHENG · 2016 to 2020
$1.7M
NIAID NIH HHS R01 AI119041NIAMS NIH HHS R01 AR069681
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

AnimalsAnimals, NewbornCell DifferentiationDEAD-box RNA HelicasesEmbryo, MammalianEpidermisFemaleIntraepithelial LymphocytesMaleMiceMice, KnockoutMicroRNAsModels, AnimalReceptors, Antigen, T-Cell, gamma-deltaReceptors, Granulocyte-Macrophage Colony-Stimulating FactorRibonuclease IIICsf1r protein, mouseDEAD-box RNA HelicasesDicer1 protein, mouseMicroRNAsReceptors, Antigen, T-Cell, gamma-deltaReceptors, Granulocyte-Macrophage Colony-Stimulating FactorRibonuclease III

Identifiers

PMID28870694
PMCPMC5837857

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.