Evidence map›Paper›PMID 28860493›Full record

ArticleScientific reports2017

CXCR3 plays a critical role for host protection against Salmonellosis.

Belal Chami, Amanda Yeung, Michael Buckland, Hongjun Liu, Genevieve M Fong, Kun Tao, Shisan Bao

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
2.6field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 26 citations in OpenAlex.

  1. Article
  2. Article
  3. Chemokine profile in the serum of patients with leptospirosis.Frontiers in cellular and infection microbiology · 2024
    Article
  4. Article
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  7. Review
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  11. Review
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  13. Article
  14. Pathogen Evasion of Chemokine Response Through Suppression of CXCL10.Frontiers in cellular and infection microbiology · 2019
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 2 countries.

Belal ChamiDepartment of Pathology, Tongren Hospital, Shanghai Jiaotong University, Shanghai, China.
Amanda YeungDiscipline of Pathology, School of Medical Sciences and Bosch Institute, The University of Sydney, Sydney, Australia.
Michael BucklandDiscipline of Pathology, School of Medical Sciences and Bosch Institute, The University of Sydney, Sydney, Australia.ORCID 0000-0003-4755-6471
Hongjun LiuDiscipline of Pathology, School of Medical Sciences and Bosch Institute, The University of Sydney, Sydney, Australia.
Genevieve M FongDiscipline of Pathology, School of Medical Sciences and Bosch Institute, The University of Sydney, Sydney, Australia.
Kun TaoDepartment of Pathology, Tongren Hospital, Shanghai Jiaotong University, Shanghai, China. taokun@shtrhospital.com.
Shisan BaoDepartment of Pathology, Tongren Hospital, Shanghai Jiaotong University, Shanghai, China. bob.bao@sydney.edu.au.
The University of Sydney · AUTongren Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

CXCR3 and its ligands are heavily associated with inflammation and have been implicated in numerous inflammatory diseases. CXCR3 plays an important role in recruiting pro-inflammatory cells, specifically neutrophils, in a model of sterile colitis whereby CXCR3

Indexed as

AnimalsCD4-Positive T-LymphocytesCecumDisease Models, AnimalGene Knockout TechniquesInterferon-gammaLiverMiceNeutrophilsReceptors, CXCR3Salmonella enteritidisSalmonella InfectionsSpleenT-Lymphocytes, Helper-InducerCxcr3 protein, mouseInterferon-gammaReceptors, CXCR3

Identifiers

PMID28860493
PMCPMC5579293
OpenAlexW2747861440

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.