Evidence map›Paper›PMID 28847826›Full record

ReviewPhilosophical transactions of the Royal Society of London. Series B, Biological sciences2017

Genome maintenance functions of the INO80 chromatin remodeller.

Ashby J Morrison

Open access · bronzeAbstract readReview
In one paragraph

Review in Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
1.6field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 35 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. DNA damage and repair: underlying mechanisms leading to microcephaly.Frontiers in cell and developmental biology · 2023
    Review
  6. Review
  7. Article
  8. Review
  9. Article
  10. Article
  11. ACTL6A promotes repair of cisplatin-induced DNA damage, a new mechanism of platinum resistance in cancer.Proceedings of the National Academy of Sciences of the United States of America · 2021
    Article
  12. Article
  13. Chromatin Remodeling and Epigenetic Regulation in Plant DNA Damage Repair.International journal of molecular sciences · 2019
    Review
  14. Article
  15. Article
  16. Article
  17. Chromatin modifiers and remodellers in DNA repair and signalling.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2017
    Article
  18. The tale of a tail: histone H4 acetylation and the repair of DNA breaks.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2017
    Review
  19. DNA repair goes hip-hop: SMARCA and CHD chromatin remodellers join the break dance.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2017
    Review
  20. The INO80 remodeller in transcription, replication and repair.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2017
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Ashby J MorrisonDepartment of Biology, Stanford University, 371 Serra Mall, Stanford, CA 94305, USA ashbym@stanford.edu.ORCID http://orcid.org/0000-0003-1228-5093
Google (United States) · US

Funding

The Role of Chromatin in Metabolic Homeostasis SupplementalR35GM119580 · NIGMS · STANFORD UNIVERSITY · PI MORRISON, ASHBY J. · 2016 to 2025
$4.7M
NIGMS NIH HHS R35 GM119580
6 · The paper itself

Abstract

Chromatin modification is conserved in all eukaryotes and is required to facilitate and regulate DNA-templated processes. For example, chromatin manipulation, such as histone post-translational modification and nucleosome positioning, play critical roles in genome stability pathways. The INO80 chromatin-remodelling complex, which regulates the abundance and positioning of nucleosomes, is particularly important for proper execution of inducible responses to DNA damage. This review discusses the participation and activity of the INO80 complex in DNA repair and cell cycle checkpoint pathways, with emphasis on the

Indexed as

Cell Cycle CheckpointsDNA RepairAtaxia Telangiectasia Mutated ProteinsDNA HelicasesSaccharomyces cerevisiaeAtaxia Telangiectasia Mutated ProteinsDNA HelicasesATM/ATRcheckpointchromatinDNA repairINO80mitosis

Identifiers

PMID28847826
PMCPMC5577467
OpenAlexW2747718030

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.