ArticleApoptosis : an international journal on programmed cell death2017

Glucotoxicity promotes aberrant activation and mislocalization of Ras-related C3 botulinum toxin substrate 1 [Rac1] and metabolic dysfunction in pancreatic islet β-cells: reversal of such metabolic defects by metformin.

Sartaj Baidwan et al.PubMed ↗Full text ↗Publisher ↗

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