Evidence map›Paper›PMID 28824923›Full record

ArticleFrontiers in cardiovascular medicine2017

Activation of Inflammatory and Pro-Thrombotic Pathways in Acute Stress Cardiomyopathy.

Timothy P Fitzgibbons, Yvonne J K Edwards, Peter Shaw, Aline Iskandar, Mohamed Ahmed, Josiah Bote, Tejen Shah, Sumita Sinha, Robert E Gerszten, John F Keaney and 2 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in cardiovascular medicine, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
0.7field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 22 citations in OpenAlex.

  1. Article
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  4. Review
  5. Article
  6. The Immunology of Takotsubo Syndrome.Frontiers in immunology · 2023
    Review
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  8. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 1 country.

Timothy P FitzgibbonsDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Yvonne J K EdwardsProgram in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Peter ShawDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Aline IskandarDivision of Cardiovascular Medicine, Maine Medical Center, Portland, ME, United States.
Mohamed AhmedDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Josiah BoteDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Tejen ShahDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Sumita SinhaDepartment of Medicine, Beth Israel Deaconess Medical Center, Boston, MA, United States.
Robert E GersztenDepartment of Medicine, Beth Israel Deaconess Medical Center, Boston, MA, United States.
John F KeaneyDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
Michael R ZileRalph H. Johnson Veterans Administration Medical Center, Medical University of South Carolina, Charleston, SC, United States.
Gerard P AurigemmaDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, United States.
University of Massachusetts Chan Medical School · USBeth Israel Deaconess Medical Center · USMaine Medical Center · USMedical University of South Carolina · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Stress cardiomyopathy (SCM) is a unique cardiac disorder that more often occurs in women. SCM presents in a similar fashion as acute myocardial infarction (AMI), with chest pain, ECG changes, and congestive heart failure. The primary distinguishing feature is the absence of thrombotic coronary occlusion in SCM. How this reduction in cardiac function occurs in the absence of coronary occlusion remains unknown. Therefore, we tested the hypothesis that a targeted proteomic comparison of patients with acute SCM and AMI might identify relevant mechanistic differences. Blood was drawn in normal controls (

Indexed as

acute myocardial infarctioncoagulationinflammationstress cardiomyopathywomen

Identifiers

PMID28824923
PMCPMC5541033
OpenAlexW2745212440

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.