Evidence map›Paper›PMID 28807260›Full record

ArticleKidney international2017

ANCA in anti-GBM disease: moving beyond a one-dimensional clinical phenotype.

Mark Canney, Mark Alan Little

Abstract readComment
PubMed Publisher
In one paragraph

Article in Kidney international, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.4field-weighted citation impact, top 33% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 10 citations in OpenAlex.

  1. [Cocaine-induced vasculitis and mimics of vasculitis].Zeitschrift fur Rheumatologie · 2023
    Review
  2. Article
  3. Macroscopic hematuria, facing an uncommon disease: Answers.Pediatric nephrology (Berlin, Germany) · 2022
    Article
  4. Article
  5. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Mark CanneyTrinity Health Kidney Centre, Trinity College Dublin, Dublin, Ireland. Electronic address: mcanney@tcd.ie.
Mark Alan LittleTrinity Health Kidney Centre, Trinity College Dublin, Dublin, Ireland.
Trinity College Dublin · IE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

McAdoo et al. propose that patients with both circulating antiglomerular basement membrane antibody and antineutrophil cytoplasm antibody demonstrate a phenotype that lies between that of single-positive antiglomerular basement membrane disease and antineutrophil cytoplasm antibody-associated vasculitis. Specifically, there may be a subset of "double-positives" that have a more favorable response to therapy. These observations, along with reports of "atypical" antiglomerular basement membrane disease, challenge us to look beyond antiglomerular basement membrane disease as a 1-dimensional entity, and better characterize its clinical spectrum.

Indexed as

Antibodies, Antineutrophil CytoplasmicAnti-Glomerular Basement Membrane DiseaseAnti-Neutrophil Cytoplasmic Antibody-Associated VasculitisAutoantibodiesBasement MembraneHumansPhenotypeAntibodies, Antineutrophil CytoplasmicAutoantibodies

Identifiers

PMID28807260
OpenAlexW2744814354

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.