Evidence map›Paper›PMID 28796976›Full record

ReviewDevelopmental period medicine

[Hfeprotein impact on iron metabolism].

Barbara Kaczorowska-Hać, Jan Jacek Kaczor

Abstract readReview
In one paragraph

Review in Developmental period medicine. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. The effect of hepcidin in rats with renal ischemia/reperfusion injury.American journal of translational research · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Barbara Kaczorowska-HaćZakład Terapii Zajęciowej, Akademia Wychowania Fizycznego i Sportu w Gdańsku, Polska.
Jan Jacek KaczorKatedra Fizjoterapii, Akademia Wychowania Fizycznego i Sportu w Gdańsku, Polska.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hereditary hemochromatosis type 1 is an autosomal recessive disorder caused by HFE gene mutations, which is an iron homeostasis metabolism controlling co-factor. Adults with male predomination present with clinical symptoms derived by iron overload in organs. The phenotype expression is individual with an influence of individual and environmental factors. Despite the fact that HFE variants are widespread, its impact still remains unknown. The article reviews the literature considering the role of HFE gene mutations regarding its impact in children.

Indexed as

ChildHemochromatosisHemochromatosis ProteinHumansIronMutationHemochromatosis ProteinHFE protein, humanIronchildrenHFE gene mutationiron

Identifiers

PMID28796976
PMCPMC8522971

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.