Evidence map›Paper›PMID 28785010›Full record

ArticleScientific reports2017

Utrophin Compensates dystrophin Loss during Mouse Spermatogenesis.

Hung-Chih Chen, Yu-Feng Chin, David J Lundy, Chung-Tiang Liang, Ya-Hui Chi, Paolin Kuo, Patrick C H Hsieh

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.1field-weighted citation impact, top 49% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 15 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Dystrophin- and Utrophin-Based Therapeutic Approaches for Treatment of Duchenne Muscular Dystrophy: A Comparative Review.BioDrugs : clinical immunotherapeutics, biopharmaceuticals and gene therapy · 2024
    Review
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 4 institutions in 1 country.

Hung-Chih ChenInstitute of Biomedical Sciences, Academia Sinica, Taipei, 115, Taiwan.
Yu-Feng ChinInstitute of Biomedical Sciences, Academia Sinica, Taipei, 115, Taiwan.
David J LundyInstitute of Biomedical Sciences, Academia Sinica, Taipei, 115, Taiwan.
Chung-Tiang LiangNational Laboratory Animal Center, National Applied Research Laboratories, Taipei, 115, Taiwan.
Ya-Hui ChiInstitute of Biotechnology and Pharmaceutical Research, National Health Research Institutes, Zhunan, Miaoli County, 350, Taiwan.ORCID 0000-0002-9216-0938
Paolin KuoDepartment of Obstetrics & Gynecology, College of Medicine, National Cheng Kung University, Tainan, 701, Taiwan.
Patrick C H HsiehInstitute of Biomedical Sciences, Academia Sinica, Taipei, 115, Taiwan. phsieh@ibms.sinica.edu.tw.ORCID 0000-0002-8910-3596
Institute of Biomedical Sciences, Academia Sinica · TWNational Cheng Kung University · TWNational Health Research Institutes · TWNational Institutes of Applied Research · TW

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Duchenne muscular dystrophy (DMD) is an X-linked genetic disorder resulting from mutations in the dystrophin gene. The mdx/utrn

Indexed as

AnimalsApoptosisCell MovementCell ProliferationDisease Models, AnimalDystrophinGene ExpressionGenotypeHaploinsufficiencyMaleMiceMice, Inbred mdxMice, KnockoutMuscle, SkeletalMuscular Dystrophy, DuchenneSpermatogenesisDystrophinUtrophin

Identifiers

PMID28785010
PMCPMC5547154
OpenAlexW2742141753

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.