ArticleFrontiers in plant science2017
Endocytosis of AtRGS1 Is Regulated by the Autophagy Pathway after D-Glucose Stimulation.
Article in Frontiers in plant science, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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Who cites it
11 citing papers in PubMed, 14 citations in OpenAlex.
- Beyond parallel pathways: molecular integration and functional crosstalk between autophagy and endocytosis in plants.Protoplasma · 2026Review
- Class VI G protein-coupled receptors in Aspergillus oryzae regulate sclerotia formation through GTPase-activating activity.Applied microbiology and biotechnology · 2024Article
- Plant Autophagy: An Intricate Process Controlled by Various Signaling Pathways.Frontiers in plant science · 2021Review
- Construction of gold-siRNARSC advances · 2020Article
- Correlation of Autophagosome Formation with Degradation and Endocytosis Arabidopsis Regulator of G-Protein Signaling (RGS1) through ATG8a.International journal of molecular sciences · 2019Article
- Linking Autophagy to Abiotic and Biotic Stress Responses.Trends in plant science · 2019Review
- Mulberry RGS negatively regulates salt stress response and tolerance.Plant signaling & behavior · 2019Article
- Glucose signaling, AtRGS1 and plant autophagy.Plant signaling & behavior · 2019Review
- Autophagy in Plants: Both a Puppet and a Puppet Master of Sugars.Frontiers in plant science · 2019Review
- Long-distance communication in Arabidopsis involving a self-activating G protein.Plant direct · 2018Article
- UDP-Glucose: A Potential Signaling Molecule in Plants?Frontiers in plant science · 2017Article
Corrections and comments
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Authors and funding
4 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Sugar, as a signal molecule, has significant functions in signal transduction in which the seven-transmembrane regulator of G-protein signaling (RGS1) protein participates. D-Glucose causes endocytosis of the AtRGS1, leading to the physical uncoupling of AtRGS1 from AtGPA1 and thus a release of the GAP activity and concomitant sustained activation of G-protein signaling. Autophagy involves in massive degradation and recycling of cytoplasmic components to survive environmental stresses. The function of autophagy in AtRGS1 endocytosis during D-glucose stimulation has not been elucidated. In this study, we investigate the relationship between autophagy and AtRGS1 in response to D-glucose. Our findings demonstrated that AtRGS1 mediated the activation of autophagy by affecting the activities of the five functional groups of protein complexes and promoted the formation of autophagosomes under D-glucose application. When the autophagy pathway was interrupted, AtRGS1 recovery increased and endocytosis of ATRGS1 was inhibited, indicating that autophagy pathway plays an important role in regulating the endocytosis and recovery of AtRGS1 after D-glucose stimulation.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.