Evidence map›Paper›PMID 28672825›Full record

ArticleMarine drugs2017

Maitotoxin Is a Potential Selective Activator of the Endogenous Transient Receptor Potential Canonical Type 1 Channel in Xenopus laevis Oocytes.

Pedro L Flores, Emma Rodríguez, Estrella Zapata, Roxana Carbó, José María Farías, Martín Martínez

Open access · goldAbstract read
In one paragraph

Article in Marine drugs, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.1field-weighted citation impact, top 24% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 14 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Pedro L FloresDepartamento de Instrumentación Electromecánica, Instituto Nacional de Cardiología "Ignacio Chávez", Juan Badiano # 1, Col. Sección XVI, México City 14080, Mexico. pelfoch07@yahoo.com.mx.
Emma RodríguezLaboratorio de Biología Celular, Departamento de Fisiología, Instituto Nacional de Cardiología "Ignacio Chávez", Juan Badiano # 1, Col. Sección XVI, México City 14080, Mexico. emarod2@yahoo.com.mx.
Estrella ZapataLaboratorio de Biología Celular, Departamento de Fisiología, Instituto Nacional de Cardiología "Ignacio Chávez", Juan Badiano # 1, Col. Sección XVI, México City 14080, Mexico. estrella_zg@yahoo.com.
Roxana CarbóDepartamento de Biomedicina Cardiovascular, Instituto Nacional de Cardiología "Ignacio Chávez", Juan Badiano # 1, Col. Sección XVI, México City 14080, Mexico. roxcarbo@gmail.com.
José María FaríasDepartamento de Fisiología, Facultad de Medicina, Universidad Nacional Autónoma de México, México City 04510, Mexico. chema_farias@hotmail.com.
Martín MartínezDepartamento de Fisiología, Instituto Nacional de Cardiología "Ignacio Chávez", Juan Badiano # 1, Col. Sección XVI, México City 14080, Mexico. martin5163@hotmail.com.
Instituto Nacional de Cardiología · MXUniversidad Nacional Autónoma de México · MX

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Maitotoxin (MTX) is the most potent marine toxin known to date. It is responsible for a particular human intoxication syndrome called

Indexed as

XenopusAnimalsElectric StimulationElectrophysiologyMarine ToxinsMembrane PotentialsOocytesOxocinsPatch-Clamp TechniquesPolyether ToxinsTRPC Cation ChannelsmaitotoxinMarine ToxinsOxocinsPolyether ToxinsTRPC Cation Channelsciguatera fish poisoningmaitotoxinTRPC channelsXenopus laevis oocytes

Identifiers

PMID28672825
PMCPMC5532640
OpenAlexW2670626680

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.