Evidence map›Paper›PMID 28653490›Full record

ReviewJournal of the European Academy of Dermatology and Venereology : JEADV2017

The role of IL-23 and the IL-23/T

G Girolomoni, R Strohal, L Puig, H Bachelez, J Barker, W H Boehncke, J C Prinz

Open access · hybridAbstract readReview
In one paragraph

Review in Journal of the European Academy of Dermatology and Venereology : JEADV, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 152 papers, 13 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
152citing papers in PubMed, 13 pooled it
9.6field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

152 citing papers in PubMed, 13 syntheses or guidelines pooled it, 282 citations in OpenAlex.

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92 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 7 institutions in 8 countries.

G GirolomoniSection of Dermatology, Department of Medicine, University of Verona, Verona, Italy.
R StrohalDepartment of Dermatology and Venerology, Federal Academic Teaching Hospital of Feldkirch, Feldkirch, Austria.
L PuigHospital de la Santa Creu i Sant Pau, Universitat Autònoma de Barcelona, Barcelona, Spain.
H BachelezSorbonne Paris Cité, Université Paris Diderot, Paris, France.
J BarkerSt John's Institute of Dermatology, King's College London, London, UK.
W H BoehnckeDivision of Dermatology, Geneva University Hospitals, Department of Pathology and Immunology, Faculty of Medicine, University of Geneva, Geneva, Switzerland.
J C PrinzDepartment of Dermatology, University of Munich, Munich, Germany.
Geneva College · USHospital de Sant Pau · ESKing's College London · GBLandeskrankenhaus Feldkirch · ATLudwig-Maximilians-Universität München · DEUniversité Paris Cité · FRUniversity of Verona · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis is a chronic, immune-mediated disease affecting more than 100 million people worldwide and up to 2.2% of the UK population. The aetiology of psoriasis is thought to originate from an interplay of genetic, environmental, infectious and lifestyle factors. The manner in which genetic and environmental factors interact to contribute to the molecular disease mechanisms has remained elusive. However, the interleukin 23 (IL-23)/T-helper 17 (T

Indexed as

Dermatologic AgentsHumansImmunity, InnateInterleukin-23PsoriasisTh17 CellsUnited KingdomDermatologic AgentsInterleukin-23

Identifiers

PMID28653490
PMCPMC5697699
OpenAlexW2730335164

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.