Expanded repertoire of

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

23 citing papers in PubMed, 44 citations in OpenAlex.

1. High-throughput microfluidic blood testing to phenotype genetically linked platelet disorders: an aid to diagnosis.Blood advances · 2023
   Article
2. FarGen: Elucidating the distribution of coding variants in the isolated population of the Faroe Islands.European journal of human genetics : EJHG · 2023
   Article
3. Hematopoietic transcription factor GFI1 promotes anchorage independence by sustaining ERK activity in cancer cells.The Journal of clinical investigation · 2022
   Article
4. CalDAG-GEFI Deficiency in a Family with Symptomatic Heterozygous and Homozygous Carriers of a Likely Pathogenic Variant inInternational journal of molecular sciences · 2021
   Article
5. Inherited Platelet Function Disorder From Novel Mutations in RAS Guanyl-Releasing Protein-2 Confirmed by Sanger Sequencing.Cureus · 2020
   Article
6. Platelet transfusion for patients with platelet dysfunction: effectiveness, mechanisms, and unanswered questions.Current opinion in hematology · 2020
   Review
7. Whole-genome sequencing of patients with rare diseases in a national health system.Nature · 2020
   Article
8. Severe bleeding and absent ADP-induced platelet aggregation associated with inherited combined CalDAG-GEFI and P2YHaematologica · 2020
   Article
9. Glanzmann thrombasthenia: genetic basis and clinical correlates.Haematologica · 2020
   Review
10. Subcellular localization of Rap1 GTPase activator CalDAG-GEFI is orchestrated by interaction of its atypical C1 domain with membrane phosphoinositides.Journal of thrombosis and haemostasis : JTH · 2020
    Article
11. RasGRP2 Structure, Function and Genetic Variants in Platelet Pathophysiology.International journal of molecular sciences · 2020
    Review
12. Molecular genetic framework underlying pulmonary arterial hypertension.Nature reviews. Cardiology · 2020
    Review
13. Impaired hemostatic activity of healthy transfused platelets in inherited and acquired platelet disorders: Mechanisms and implications.Science translational medicine · 2019
    Article
14. Diagnostic high-throughput sequencing of 2396 patients with bleeding, thrombotic, and platelet disorders.Blood · 2019
    Article
15. The inhibition of Bax activation-induced apoptosis by RasGRP2 via R-Ras-PI3K-Akt signaling pathway in the endothelial cells.Scientific reports · 2019
    Article
16. Hereditary platelet function disorder fromHaematologica · 2019
    Article
17. RAP GTPases and platelet integrin signaling.Platelets · 2019
    Review
18. Functional redundancy between RAP1 isoforms in murine platelet production and function.Blood · 2018
    Article
19. [The clinical characteristics and molecular pathogenesis of a variant Glanzmann's thrombasthenia-like pedigree].Zhonghua xue ye xue za zhi = Zhonghua xueyexue zazhi · 2018
    Article
20. Functional Genomics for the Identification of Modulators of Platelet-Dependent Thrombus Formation.TH open : companion journal to thrombosis and haemostasis · 2018
    Review

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

Authors and funding

22 authors at 11 institutions in 6 countries.

Funding

Abstract

Indexed as