Evidence map›Paper›PMID 28612294›Full record

ReviewNeurotoxicity research2018

The Potential Role of BMAA in Neurodegeneration.

Tracie Caller, Patricia Henegan, Elijah Stommel

Abstract readReview
PubMed Publisher
In one paragraph

Review in Neurotoxicity research, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed, 1 pooled it
2.1field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 1 synthesis or guideline pooled it, 27 citations in OpenAlex.

  1. Pooled it
  2. Artificial Intelligence in The Management of Neurodegenerative Disorders.CNS & neurological disorders drug targets · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Tracie CallerDepartment of Neurology, Dartmouth-Hitchcock Medical Center, Lebanon, NH, 03756, USA. tracie.a.caller@dartmouth.edu.
Patricia HeneganDepartment of Neurology, Dartmouth-Hitchcock Medical Center, Lebanon, NH, 03756, USA.
Elijah StommelDepartment of Neurology, Dartmouth-Hitchcock Medical Center, Lebanon, NH, 03756, USA.
Dartmouth–Hitchcock Medical Center · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Neurodegenerative diseases are a major public health issue throughout the world with devastating effects on patients and families. Sporadic forms of neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease, and amyotrophic lateral sclerosis are generally thought to develop as a consequence of genetic susceptibility and environmental influences. A number of environmental triggers have been identified in association with amyotrophic lateral sclerosis and Parkinson's disease. We discuss the role of β-methylamino-L-alanine in the development of neurodegeneration and the potential importance of this neurotoxin as a risk for neurodegeneration.

Indexed as

Amino Acids, DiaminoAnimalsCyanobacteria ToxinsGene-Environment InteractionHumansNeurodegenerative DiseasesNeurotoxinsRisk FactorsAmino Acids, Diaminobeta-N-methylamino-L-alanineCyanobacteria ToxinsNeurotoxinsAlzheimer’s diseaseAmyotrophic lateral sclerosisNeurodegenerationParkinson’s diseaseβ-methylamino-L-alanine

Identifiers

PMID28612294
OpenAlexW2624792738

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.