Evidence map›Paper›PMID 28572456›Full record

ReviewCold Spring Harbor perspectives in biology2017

Intracellular Motility of Intermediate Filaments.

Rudolf E Leube, Marcin Moch, Reinhard Windoffer

Open access · bronzeAbstract readReview
In one paragraph

Review in Cold Spring Harbor perspectives in biology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Assembly of NFL and desmin intermediate filaments: Headed in the right direction.Proceedings of the National Academy of Sciences of the United States of America · 2021
    Article
  6. Hemidesmosome-Related Keratin Filament Bundling and Nucleation.International journal of molecular sciences · 2021
    Article
  7. Article
  8. Article
  9. Article
  10. Review
  11. Motor Proteins.Cold Spring Harbor perspectives in biology · 2018
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Rudolf E LeubeInstitute of Molecular and Cellular Anatomy, RWTH Aachen University, 52074 Aachen, Germany.
Marcin MochInstitute of Molecular and Cellular Anatomy, RWTH Aachen University, 52074 Aachen, Germany.
Reinhard WindofferInstitute of Molecular and Cellular Anatomy, RWTH Aachen University, 52074 Aachen, Germany.
RWTH Aachen University · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

SUMMARYThe establishment and continuous cell type-specific adaptation of cytoplasmic intermediate filament (IF) networks are linked to various types of IF motility. Motor protein-driven active transport, linkage to other cellular structures, diffusion of small soluble subunits, and intrinsic network elasticity all contribute to the motile behavior of IFs. These processes are subject to regulation by multiple signaling pathways. IF motility is thereby connected to and involved in many basic cellular processes guarding the maintenance of cell and tissue integrity. Disturbances of IF motility are linked to diseases that are characterized by cytoplasmic aggregates containing IF proteins together with other cellular components.

Indexed as

Actin CytoskeletonActinsAnimalsAxonsCell Line, TumorCell MovementCytoplasmDiffusionGreen Fluorescent ProteinsHomeostasisHumansIntermediate Filament ProteinsIntermediate FilamentsKeratinsMicroscopy, ConfocalMicrotubulesActinsGreen Fluorescent ProteinsIntermediate Filament ProteinsKeratins

Identifiers

PMID28572456
PMCPMC5453390
OpenAlexW2620951490

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.