Evidence map›Paper›PMID 28561888›Full record

ArticleDevelopmental psychobiology2017

Affiliation with substance-using peers: Examining gene-environment correlations among parent monitoring, polygenic risk, and children's impulsivity.

Kit K Elam, Laurie Chassin, Kathryn Lemery-Chalfant, Danielle Pandika, Frances L Wang, Kaitlin Bountress, Danielle Dick, Arpana Agrawal

Abstract read
In one paragraph

Article in Developmental psychobiology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed.

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  8. A gene-environment cascade theoretical framework of developmental psychopathology.Journal of psychopathology and clinical science · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Kit K ElamT. Denny Sanford School of Social and Family Dynamics, Arizona State University, Tempe, Arizona.ORCID 0000-0003-0319-5897
Laurie ChassinDepartment of Psychology, Arizona State University, Tempe, Arizona.
Kathryn Lemery-ChalfantDepartment of Psychology, Arizona State University, Tempe, Arizona.
Danielle PandikaDepartment of Psychology, Arizona State University, Tempe, Arizona.
Frances L WangDepartment of Psychiatry, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania.
Kaitlin BountressNational Crime Victims Research & Treatment Center, Medical University of South Carolina, Charleston, South Carolina.
Danielle DickDepartment of Psychology, Virginia Commonwealth University, Richmond, Virginia.
Arpana AgrawalDepartment of Psychological & Brain Sciences, Washington University in St. Louis, Saint Louis, Missouri.

Funding

PILOT--GENETIC EPIDEMIOLOGY OF ALCOHOL AND TOBACCO CONSUMPTION IN CHINESE SAMPLEP50AA011998 · NIAAA · WASHINGTON UNIVERSITY · PI SLUTSKE, WENDY S · 1999 to 2008
$12.6M
Resource CoreP60AA011998 · NIAAA · WASHINGTON UNIVERSITY · PI HEATH, ANDREW C. · 2009 to 2013
$8.8M
Training in Child Mental Health/Primary PreventionT32MH018387 · NIMH · ARIZONA STATE UNIVERSITY-TEMPE CAMPUS · PI CHASSIN, LAURIE A · 1987 to 2014
$3.8M
Substance Use Among Children of AlcoholicsR01AA016213 · NIAAA · ARIZONA STATE UNIVERSITY-TEMPE CAMPUS · PI CHASSIN, LAURIE A · 2006 to 2010
$3.1M
Exploring a novel approach to clarify parenting effects on drinking outcomesR21AA022097 · NIAAA · ARIZONA STATE UNIVERSITY-TEMPE CAMPUS · PI CHASSIN, LAURIE A · 2013 to 2014
$382k
Serotonin Genes and Adolescent Alcohol and Drug Involvement:Mechanisms of RiskF31AA023128 · NIAAA · ARIZONA STATE UNIVERSITY-TEMPE CAMPUS · PI WANG, FRANCES L · 2014 to 2016
$108k
Parent and Peer Influences on Alcohol Use Problems: A Genetically Informed StudyF31AA021612 · NIAAA · ARIZONA STATE UNIVERSITY-TEMPE CAMPUS · PI BOUNTRESS, KAITLIN ELIZABETH · 2012 to 2013
$75k
NIAAA NIH HHS F31 AA021612NIAAA NIH HHS F31 AA023128NIAAA NIH HHS P50 AA011998NIAAA NIH HHS P60 AA011998NIAAA NIH HHS R01 AA016213NIAAA NIH HHS R21 AA022097NIMH NIH HHS T32 MH018387
6 · The paper itself

Abstract

Parental monitoring can buffer the effect of deviant peers on adolescents' substance use by reducing affiliation with substance-using peers. However, children's genetic predispositions may evoke poorer monitoring, contributing to negative child outcomes. We examined evocative genotype-environment correlations underlying children's genetic predisposition for behavioral undercontrol and parental monitoring in early adolescence via children's impulsivity in middle childhood, and the influence of parental monitoring on affiliation with substance-using peers a year and a half later (n = 359). Genetic predisposition for behavioral undercontrol was captured using a polygenic risk score, and a portion of passive rGE was controlled by including parents' polygenic risk scores. Children's polygenic risk predicted poorer parental monitoring via greater children's impulsivity, indicating evocative rGE, controlling for a portion of passive rGE. Poorer parental monitoring predicted greater children's affiliation with substance-using peers a year and a half later. Results are discussed with respect to gene-environment correlations within developmental cascades.

Indexed as

Gene-Environment InteractionGenetic Predisposition to DiseasePeer GroupAdolescentAdolescent BehaviorChildFemaleHumansImpulsive BehaviorLongitudinal StudiesMaleParentingParentsRisk FactorsSocial BehaviorSocial Environmentdeviant peersgene-environment correlationimpulsivityparent monitoringpolygenic risk score

Identifiers

PMID28561888
PMCPMC6035731

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.