Evidence map›Paper›PMID 28551112›Full record

ArticleAppetite2017

Resting state hypothalamic response to glucose predicts glucose-induced attenuation in the ventral striatal response to food cues.

Shan Luo, A James Melrose, Hilary Dorton, Jasmin Alves, John R Monterosso, Kathleen A Page

Open access · greenAbstract read
In one paragraph

Article in Appetite, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.5field-weighted citation impact, top 42% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 12 citations in OpenAlex.

  1. Trial
  2. Article
  3. Review
  4. Review
  5. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Shan LuoDivision of Endocrinology, Keck School of Medicine, University of Southern California, 90089, United States; Department of Psychology, University of Southern California, 90089, United States; Diabetes and Obesity Research Institute, Keck School of Medicine, University of Southern California, 90089, United States.
A James MelroseDepartment of Psychology, University of Southern California, 90089, United States; Diabetes and Obesity Research Institute, Keck School of Medicine, University of Southern California, 90089, United States.
Hilary DortonNeuroscience Graduate Program, University of Southern California, 90089, United States; Diabetes and Obesity Research Institute, Keck School of Medicine, University of Southern California, 90089, United States.
Jasmin AlvesDiabetes and Obesity Research Institute, Keck School of Medicine, University of Southern California, 90089, United States.
John R MonterossoDepartment of Psychology, University of Southern California, 90089, United States; Neuroscience Graduate Program, University of Southern California, 90089, United States.
Kathleen A PageDivision of Endocrinology, Keck School of Medicine, University of Southern California, 90089, United States; Neuroscience Graduate Program, University of Southern California, 90089, United States; Diabetes and Obesity Research Institute, Keck School of Medicine, University of Southern California, 90089, United States. Electronic address: kpage@usc.edu.
University of Southern California · US

Funding

Neural Mechanisms for Appetitive Responses to High Reward FoodsR01DK102794 · NIDDK · UNIVERSITY OF SOUTHERN CALIFORNIA · PI PAGE, KATHLEEN ALANNA · 2016 to 2020
$2.7M
NIDDK NIH HHS R01 DK102794
6 · The paper itself

Abstract

Feeding behavior is regulated by a complex interaction of central nervous system responses to metabolic signals that reflect nutrient availability and to food cues that trigger appetitive responses. Prior work has shown that the hypothalamus is a key brain area that senses and responds to changes in metabolic signals, and exposure to food cues induces the activation of brain areas involved in reward processing. However, it is not known how the hypothalamic responses to changes in metabolic state are related to reward responses to food cues. This study aimed to understand whether changes in hypothalamic activity in response to glucose-induced metabolic signals are linked to food-cue reactivity within brain areas involved in reward processing. We combined two neuroimaging modalities (Arterial Spin Labeling and Blood Oxygen Level Dependent) to measure glucose-induced changes in hypothalamic cerebral blood flow (CBF) and food-cue task induced changes in brain activity within reward-related regions. Twenty-five participants underwent a MRI session following glucose ingestion and a subset of twenty individuals underwent an additional water session on a separate day as a control condition (drink order randomized). Hunger was assessed before and after drink consumption. We observed that individuals who had a greater reduction in hypothalamic CBF exhibited a greater reduction in left ventral striatum food cue reactivity (Spearman's rho = 0.46, P = 0.048) following glucose vs. water ingestion. These results are the first to use multimodal imaging to demonstrate a link between hypothalamic metabolic signaling and ventral striatal food cue reactivity.

Indexed as

CuesAdultAppetiteBody Mass IndexDietFemaleGlucoseHumansHungerHypothalamusMagnetic Resonance ImagingMaleNeuroimagingPhotic StimulationRewardYoung AdultGlucoseAppetite regulationBrainEating behaviorGlucose

Identifiers

PMID28551112
PMCPMC5557629
OpenAlexW2619818880

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.