Evidence map›Paper›PMID 28542314›Full record

ArticlePloS one2017

Toward a more personalized motor function rehabilitation in Myotonic dystrophy type 1: The role of neuroplasticity.

Simona Portaro, Antonino Naro, Antonino Chillura, Luana Billeri, Alessia Bramanti, Placido Bramanti, Carmelo Rodolico, Rocco Salvatore Calabrò

Abstract read
In one paragraph

Article in PloS one, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. Article
  8. Article
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Simona PortaroIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.
Antonino NaroIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.
Antonino ChilluraIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.
Luana BilleriIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.
Alessia BramantiDepartment of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Placido BramantiIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.
Carmelo RodolicoInstitute of Applied Sciences and Intelligent Systems "ISASI Edoardo Caianello", National Research Council of Italy, Messina, Italy.
Rocco Salvatore CalabròIstitute of Scientific Research and Care "IRCCS Centro Neurolesi Bonino-Pulejo", Messina, Italy.ORCID http://orcid.org/0000-0002-8566-3166

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Myotonic dystrophy type 1 (DM1) is the most prevalent adult muscular dystrophy, often accompanied by impairments in attention, memory, visuospatial and executive functions. Given that DM1 is a multi-system disorder, it requires a multi-disciplinary approach, including effective rehabilitation programs, focusing on the central nervous system neuroplasticity, in order to develop patient-tailored rehabilitative procedures for motor function recovery. Herein, we performed a transcranial magnetic stimulation (TMS) study aimed at investigating central motor conduction time, sensory-motor plasticity, and cortical excitability in 7 genetically defined DM1 patients. As compared to healthy individuals, DM1 patients showed a delayed central motor conduction time and an abnormal sensory-motor plasticity, with no alteration of cortical excitability. These findings may be useful to define patient-tailored motor rehabilitative programs.

Indexed as

AdolescentAdultAgedCentral Nervous SystemElectromyographyFemaleHumansMaleMemoryMiddle AgedMyotonic DystrophyNeuritisNeuronal PlasticityRecovery of FunctionSensorimotor CortexTranscranial Magnetic Stimulation

Identifiers

PMID28542314
PMCPMC5444819

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.