ArticleFrontiers in plant science2017
Genotype-Dependent Interaction of Lentil Lines with
Article in Frontiers in plant science, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
10 citing papers in PubMed.
- Evaluation of Lens culinaris germplasm against ascochyta blight by applying phenotyping and defense genes attributes.Scientific reports · 2025Article
- Genetic mapping of Ascochyta blight resistance in an ILL6002 × Indianhead lentil mapping population.The plant genome · 2025Article
- Expression Analysis of theGenes · 2022Article
- Breeding and Genomics Interventions for Developing Ascochyta Blight Resistant Grain Legumes.International journal of molecular sciences · 2022Review
- Ingenious characterization and assessment of lentil germplasm collection to aphidFrontiers in plant science · 2022Article
- Genotype-Specific Expression and NLR Repertoire Contribute to Phenotypic Resistance Diversity inFrontiers in plant science · 2021Article
- Interactive Gene Expression Patterns of Susceptible and ResistantFrontiers in microbiology · 2020Article
- Using a transcriptome sequencing approach to explore candidate resistance genes against stemphylium blight in the wild lentil species Lens ervoides.BMC plant biology · 2019Article
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Ascochyta blight of lentil is a prevalent disease in many lentil producing regions and can cause major yield and grain quality losses. The most environmentally acceptable and economically profitable method of control is to develop varieties with high levels of durable resistance. Genetic studies to date suggest that ascochyta blight resistance genes (R-gene) in lentil lines CDC Robin, ILL 7537, 964a-46, and ILL 1704 are non-allelic. To understand how different R-genes manifest resistance in these genotypes and an accession of
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