Evidence map›Paper›PMID 28336819›Full record

ArticlePhysiological reports2017

Fundamental hemodynamic mechanisms mediating the response to myocardial ischemia in conscious paraplegic mice: cardiac output versus peripheral resistance.

Heidi L Lujan, Stephen E DiCarlo

Open access · goldAbstract read
In one paragraph

Article in Physiological reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.9field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Chronic, complete cervicalJournal of applied physiology (Bethesda, Md. : 1985) · 2018
    Article
  5. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Heidi L LujanDepartment of Physiology, Wayne State University School of Medicine, Detroit, Michigan.
Stephen E DiCarloDepartment of Physiology, Wayne State University School of Medicine, Detroit, Michigan sdicarlo@med.wayne.edu.
Wayne State University · US

Funding

Vagal Plasticity after Cord Injury Exacerbates Post-Infarct Cardiac RemodelingR01HL122223 · NHLBI · WAYNE STATE UNIVERSITY · PI DICARLO, STEPHEN E · 2015 to 2018
$1.5M
NHLBI NIH HHS R01 HL122223
6 · The paper itself

Abstract

Autonomic dysfunction, a relative sedentary lifestyle, a reduced muscle mass and increased adiposity leads to metabolic abnormalities that accelerate the development of coronary artery disease (CAD) in individuals living with spinal cord injury (SCI). An untoward cardiac incident is related to the degree of CAD, suggesting that the occurrence of a significant cardiac event is significantly higher for individuals with SCI Thus, understanding the fundamental hemodynamic mechanisms mediating the response to myocardial ischemia has the potential to positively impact individuals and families living with SCI Accordingly, we systematically investigated if thoracic level 5 spinal cord transection (T

Indexed as

AnimalsArterial PressureCardiac OutputHeartHeart RateHemodynamicsMaleMiceMyocardial IschemiaParaplegiaStroke VolumeVascular ResistanceCoronary artery diseasemyocardial ischemiaspinal reflex

Identifiers

PMID28336819
PMCPMC5371571
OpenAlexW2599530272

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.