Evidence map›Paper›PMID 28320735›Full record

ArticleThe EMBO journal2017

The retinoblastoma homolog RBR1 mediates localization of the repair protein RAD51 to DNA lesions in

Sascha Biedermann, Hirofumi Harashima, Poyu Chen, Maren Heese, Daniel Bouyer, Kostika Sofroni, Arp Schnittger

Open access · bronzeAbstract read
In one paragraph

Article in The EMBO journal, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 47 papers.

0numbers the graph read from it
0cells of the map it votes in
47citing papers in PubMed
3.2field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

47 citing papers in PubMed, 88 citations in OpenAlex.

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  12. Sugar signals pedal the cell cycle!Frontiers in plant science · 2024
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 3 countries.

Sascha BiedermannDepartment of Molecular Mechanisms of Phenotypic Plasticity, Institut de Biologie Moléculaire des Plantes du Centre National de la Recherche Scientifique, Université de Strasbourg, Strasbourg, France.
Hirofumi HarashimaRIKEN Center for Sustainable Resource Science, Tsurumi, Yokohama, Japan.ORCID 0000-0003-3370-4111
Poyu ChenDepartment of Developmental Biology, Biozentrum Klein Flottbek University of Hamburg, Hamburg, Germany.
Maren HeeseDepartment of Developmental Biology, Biozentrum Klein Flottbek University of Hamburg, Hamburg, Germany.ORCID 0000-0002-1070-9107
Daniel BouyerInstitut de Biologie de l'Ecole Normale Supérieure, CNRS UMR 8197-INSERM U 1024, Paris, France.
Kostika SofroniDepartment of Developmental Biology, Biozentrum Klein Flottbek University of Hamburg, Hamburg, Germany.
Arp SchnittgerDepartment of Molecular Mechanisms of Phenotypic Plasticity, Institut de Biologie Moléculaire des Plantes du Centre National de la Recherche Scientifique, Université de Strasbourg, Strasbourg, France arp.schnittger@uni-hamburg.de.ORCID 0000-0001-7067-0091
Universität Hamburg · DECentre National de la Recherche Scientifique · FRRIKEN Center for Sustainable Resource Science · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The retinoblastoma protein (Rb), which typically functions as a transcriptional repressor of E2F-regulated genes, represents a major control hub of the cell cycle. Here, we show that loss of the

Indexed as

DNA RepairArabidopsisArabidopsis ProteinsCell DeathDNA, PlantGene DeletionProtein BindingRad51 RecombinaseArabidopsis ProteinsATRAD51 protein, ArabidopsisDNA, PlantRad51 RecombinaseRBR1 protein, ArabidopsisArabidopsisDNA‐damage responseRAD51retinoblastoma

Identifiers

PMID28320735
PMCPMC5412766
OpenAlexW2600254018

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.