Evidence map›Paper›PMID 28317021›Full record

ArticleeNeuro

The BLOC-1 Subunit Pallidin Facilitates Activity-Dependent Synaptic Vesicle Recycling.

Xun Chen, Wenpei Ma, Shixing Zhang, Jeremy Paluch, Wanlin Guo, Dion K Dickman

Open access · goldAbstract read
In one paragraph

Article in eNeuro. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 30 papers.

0numbers the graph read from it
0cells of the map it votes in
30citing papers in PubMed
3.5field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

30 citing papers in PubMed, 49 citations in OpenAlex.

  1. Genetic dissection of theMolecular biology of the cell · 2025
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  9. Tissue-specific knockout inbioRxiv : the preprint server for biology · 2023
    Article
  10. Physiological and Pathological Roles of the Cytohesin Family in Neurons.International journal of molecular sciences · 2022
    Review
  11. Review
  12. Article
  13. Article
  14. The auxiliary glutamate receptor subunit dSol-1 promotes presynaptic neurotransmitter release and homeostatic potentiation.Proceedings of the National Academy of Sciences of the United States of America · 2020
    Article
  15. Developmental arrest ofDevelopment (Cambridge, England) · 2020
    Article
  16. A Screen for Synaptic Growth Mutants Reveals Mechanisms That Stabilize Synaptic Strength.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2019
    Article
  17. Article
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  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Xun ChenDepartment of Biological Sciences, University of Southern California, Los Angeles, CA 90089; Neuroscience Graduate Program, University of Southern California, Los Angeles, CA 90089.ORCID 0000-0001-6871-2758
Wenpei MaDepartment of Biological Sciences, University of Southern California , Los Angeles, CA 90089.
Shixing ZhangDepartment of Biological Sciences, University of Southern California , Los Angeles, CA 90089.
Jeremy PaluchDepartment of Biological Sciences, University of Southern California , Los Angeles, CA 90089.
Wanlin GuoDepartment of Biological Sciences, University of Southern California , Los Angeles, CA 90089.
Dion K DickmanDepartment of Biological Sciences, University of Southern California , Los Angeles, CA 90089.ORCID 0000-0003-1884-284X
University of Southern California · US

Funding

Molecular Mechanisms Governing the Homeostatic Control of Synaptic StrengthR01NS091546 · NINDS · UNIVERSITY OF SOUTHERN CALIFORNIA · PI DICKMAN, DION KAI · 2015 to 2024
$4.3M
Dysbindin and the Mechanisms Controlling Homeostatic Synaptic PlasticityR00MH092351 · NIMH · UNIVERSITY OF SOUTHERN CALIFORNIA · PI DICKMAN, DION KAI · 2012 to 2014
$696k
NIMH NIH HHS R00 MH092351NINDS NIH HHS R01 NS091546
6 · The paper itself

Abstract

Membrane trafficking pathways must be exquisitely coordinated at synaptic terminals to maintain functionality, particularly during conditions of high activity. We have generated null mutations in the

Indexed as

AnimalsAnimals, Genetically ModifiedDrosophilaDrosophila ProteinsDysbindinDystrophin-Associated ProteinsEndocytosisEndosomesEye ProteinsHomeostasisImmunoblottingImmunohistochemistryMicroscopy, ElectronMicrotubule-Associated ProteinsMicrotubulesNeuromuscular JunctionBLOS1 protein, DrosophilaDrosophila ProteinsDysbindinDysb protein, DrosophilaDystrophin-Associated ProteinsEye ProteinsMicrotubule-Associated ProteinsPldn protein, DrosophilaBLOC-1Drosophilaendocytosisendosomeneuromuscular junctionsynaptic vesicle

Identifiers

PMID28317021
PMCPMC5356223
OpenAlexW2580006739

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.