Evidence map›Paper›PMID 28257081›Full record

ReviewNutrients2017

Matching Meals to Body Clocks-Impact on Weight and Glucose Metabolism.

Amy T Hutchison, Gary A Wittert, Leonie K Heilbronn

Open access · goldAbstract readReview
In one paragraph

Review in Nutrients, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 1 pooled it
2.2field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 1 synthesis or guideline pooled it, 35 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. [Live combinedNan fang yi ke da xue xue bao = Journal of Southern Medical University · 2025
    Article
  4. Article
  5. Chrono-Nutrition: Circadian Rhythm and Personalized Nutrition.International journal of molecular sciences · 2023
    Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Review
  12. Article
  13. Article
  14. Article
  15. Review
  16. Social dynamics modeling of chrono-nutrition.PLoS computational biology · 2019
    Article
  17. Review
  18. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Amy T HutchisonAdelaide Medical School, The University of Adelaide, Adelaide SA 5000, Australia. amy.hutchison@adelaide.edu.au.
Gary A WittertAdelaide Medical School, The University of Adelaide, Adelaide SA 5000, Australia. gary.wittert@adelaide.edu.au.
Leonie K HeilbronnAdelaide Medical School, The University of Adelaide, Adelaide SA 5000, Australia. leonie.heilbronn@adelaide.edu.au.
South Australian Health and Medical Research Institute · AU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The prevalence of type 2 diabetes continues to rise worldwide and is reaching pandemic proportions. The notion that this is due to obesity, resulting from excessive energy consumption and reduced physical activity, is overly simplistic. Circadian de-synchrony, which occurs when physiological processes are at odds with timing imposed by internal clocks, also promotes obesity and impairs glucose tolerance in mouse models, and is a feature of modern human lifestyles. The purpose of this review is to highlight what is known about glucose metabolism in animal and human models of circadian de-synchrony and examine the evidence as to whether shifts in meal timing contribute to impairments in glucose metabolism, gut hormone secretion and the risk of type 2 diabetes. Lastly, we examine whether restricting food intake to discrete time periods, will prevent or reverse abnormalities in glucose metabolism with the view to improving metabolic health in shift workers and in those more generally at risk of chronic diseases such as type 2 diabetes and cardiovascular disease.

Indexed as

Body WeightCircadian RhythmMealsAnimalsBlood GlucoseCarbohydrate MetabolismDiabetes Mellitus, Type 2DietDisease Models, AnimalExerciseGastrointestinal HormonesHumansLife StyleObesityPrevalenceRandomized Controlled Trials as TopicBlood GlucoseGastrointestinal Hormoneschronic disease riskcircadian rhythmglucose metabolismtime-restricted feedingtype 2 diabetes

Identifiers

PMID28257081
PMCPMC5372885
OpenAlexW2593643084

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.