ReviewPhilosophical transactions of the Royal Society of London. Series B, Biological sciences2017
Neuronal control of fixation and fixational eye movements.
Review in Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 110 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
110 citing papers in PubMed, 229 citations in OpenAlex.
- Loss of neuronal population organization links pathology to behavior in a model of Alzheimer's disease.Proceedings of the National Academy of Sciences of the United States of America · 2026Article
- Pre-stimulus microsaccadic inhibition reflects both temporal and sensory expectations.Experimental brain research · 2026Article
- Eye movements and ocular biomarkers in mild traumatic brain injury: from oculomotor pathophysiology to diagnosis and prognosis.Journal of neurology · 2026Review
- Fixation Task: A Simple Eye Movement Task Reveals an Impairment in Schizophrenia.Schizophrenia bulletin · 2026Article
- Amblyopia Severity Predicts Fixation Dysfunction Independent of Nystagmus and Foveation Disruption in Fusion Maldevelopment Nystagmus.Investigative ophthalmology & visual science · 2026Article
- Frequency-dependent modulation of foveal contrast sensitivity by fine-scale exogenously triggered attention.eLife · 2026Article
- Digital measurement of ocular microtremor in Parkinson's Disease: Analytical and clinical validation.PLOS digital health · 2026Article
- An Exploratory Study of the Relationship Between Phoria, Oculomotor Skills and Visual Symptoms in Children Aged 5 to 8 Years.Journal of eye movement research · 2026Article
- Loss of neuronal population organization links pathology to behavior in a model of Alzheimer's disease.bioRxiv : the preprint server for biology · 2026Article
- Eye tracking as a digital biomarker in neurodegenerative diseases.Journal of neurology · 2026Review
- Physiological basis of resolution acuity in vision.Nature communications · 2026Article
- Systematic arcminute-scale fixational offsets in patients with early visual cortex damage.Journal of vision · 2026Article
- Early screening of mild cognitive impairment based on eye-tracking technology: a cross-sectional study.Aging clinical and experimental research · 2026Article
- Perceptual punctuation: fixational eye movements reveal segmentation of auditory streams.Frontiers in neuroscience · 2026Article
- Chronic sleep restriction induces oculomotor impairment with inadequate compensation.Sleep advances : a journal of the Sleep Research Society · 2026Article
- The effects of unilateral deprivation amblyopia on fixation stability.Frontiers in neuroscience · 2026Article
- Detection of abnormal cortex in patients with orbital fractures.Scientific reports · 2025Article
- Lateralized visuotopic organization in the macaque superior colliculus revealed by fMRI.Progress in neurobiology · 2025Article
- Oculomotor Control in Preterm Infants: Insights from Eye-Tracking Technology.Journal of clinical medicine · 2025Article
- The Impact of Bulbar and Upper Motor Neuron Involvement on Oculomotor Movement in Amyotrophic Lateral Sclerosis.Brain and behavior · 2025Article
50 more citing papers are in PubMed but not listed here.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 3 institutions in 3 countries.
Funding
Abstract
Ocular fixation is a dynamic process that is actively controlled by many of the same brain structures involved in the control of eye movements, including the superior colliculus, cerebellum and reticular formation. In this article, we review several aspects of this active control. First, the decision to move the eyes not only depends on target-related signals from the peripheral visual field, but also on signals from the currently fixated target at the fovea, and involves mechanisms that are shared between saccades and smooth pursuit. Second, eye position during fixation is actively controlled and depends on bilateral activity in the superior colliculi and medio-posterior cerebellum; disruption of activity in these circuits causes systematic deviations in eye position during both fixation and smooth pursuit eye movements. Third, the eyes are not completely still during fixation but make continuous miniature movements, including ocular drift and microsaccades, which are controlled by the same neuronal mechanisms that generate larger saccades. Finally, fixational eye movements have large effects on visual perception. Ocular drift transforms the visual input in ways that increase spatial acuity; microsaccades not only improve vision by relocating the fovea but also cause momentary changes in vision analogous to those caused by larger saccades.This article is part of the themed issue 'Movement suppression: brain mechanisms for stopping and stillness'.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.