Evidence map›Paper›PMID 28166682›Full record

ReviewAutoimmunity2017

Clonally expanded cytotoxic CD4

Hamid Mattoo, John H Stone, Shiv Pillai

Open access · greenAbstract readReview
In one paragraph

Review in Autoimmunity, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 44 papers.

0numbers the graph read from it
0cells of the map it votes in
44citing papers in PubMed
8.4field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

44 citing papers in PubMed, 115 citations in OpenAlex.

  1. Review
  2. Article
  3. Cytotoxic T Cells: Kill, Memorize, and Mask to Maintain Immune Homeostasis.International journal of molecular sciences · 2025
    Review
  4. Article
  5. Article
  6. Review
  7. Article
  8. Article
  9. Article
  10. Article
  11. Review
  12. Article
  13. Immune dysregulation in immunoglobulin G4-related disease.The Japanese dental science review · 2023
    Review
  14. Article
  15. Thoracic manifestations of IgG4-related disease.Respirology (Carlton, Vic.) · 2023
    Review
  16. Article
  17. Review
  18. Review
  19. Article
  20. [Immunoglobulin-G4-related disease].Zeitschrift fur Rheumatologie · 2022
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Hamid Mattooa Massachusetts General Hospital, Harvard Medical School , Boston , MA , USA.
John H Stonea Massachusetts General Hospital, Harvard Medical School , Boston , MA , USA.
Shiv Pillaia Massachusetts General Hospital, Harvard Medical School , Boston , MA , USA.
Massachusetts General Hospital · USHarvard University · US

Funding

The importance of the IgG4 glycome in IgG4-related disease(s)U19AI110495 · NIAID · MASSACHUSETTS GENERAL HOSPITAL · PI Ramnik J Xavier · 2014 to 2026
$7.9M
NIAID NIH HHS U19 AI110495
6 · The paper itself

Abstract

IgG4-related disease (IgG4-RD) is a systemic condition of unknown cause characterized by highly fibrotic lesions, with dense lymphoplasmacytic infiltrates containing a preponderance of IgG4-expressing plasma cells. CD4

Indexed as

Cytotoxicity, ImmunologicDisease SusceptibilityAnimalsB-LymphocytesCD4-Positive T-LymphocytesCell CommunicationClonal EvolutionCytokinesFibrosisHumansImmunoglobulin Class SwitchingImmunoglobulin GLymphocyte ActivationT-Lymphocyte SubsetsCytokinesImmunoglobulin GCD4+ CTLfibrosisIgG4-RDlymphoplasmacytic infiltrate

Identifiers

PMID28166682
PMCPMC5880292
OpenAlexW2587213644

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.