ArticleBehavior genetics2017
Genetic and Environmental Structure of DSM-IV Criteria for Antisocial Personality Disorder: A Twin Study.
Article in Behavior genetics, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
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Who cites it
8 citing papers in PubMed.
- Case Studies on Dissocial Personality-Bad or Ill?Healthcare (Basel, Switzerland) · 2024Article
- Update on Antisocial Personality Disorder.Current psychiatry reports · 2024Review
- Aggression, Aggression-Related Psychopathologies and Their Models.Frontiers in behavioral neuroscience · 2022Review
- Redefining phenotypes to advance psychiatric genetics: Implications from hierarchical taxonomy of psychopathology.Journal of abnormal psychology · 2020Review
- A Psychometric Evaluation of theAssessment · 2020Article
- A Hierarchical Taxonomy of Psychopathology Can Transform Mental Health Research.Perspectives on psychological science : a journal of the Association for Psychological Science · 2019Review
- Genetically Informative Mediation Modeling Applied to Stressors and Personality-Disorder Traits in Etiology of Alcohol Use Disorder.Behavior genetics · 2019Article
- Prediction of alcohol use disorder using personality disorder traits: a twin study.Addiction (Abingdon, England) · 2018Observational
Corrections and comments
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Authors and funding
9 authors.
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Abstract
Results from previous studies on DSM-IV and DSM-5 Antisocial Personality Disorder (ASPD) have suggested that the construct is etiologically multidimensional. To our knowledge, however, the structure of genetic and environmental influences in ASPD has not been examined using an appropriate range of biometric models and diagnostic interviews. The 7 ASPD criteria (section A) were assessed in a population-based sample of 2794 Norwegian twins by a structured interview for DSM-IV personality disorders. Exploratory analyses were conducted at the phenotypic level. Multivariate biometric models, including both independent and common pathways, were compared. A single phenotypic factor was found, and the best-fitting biometric model was a single-factor common pathway model, with common-factor heritability of 51% (95% CI 40-67%). In other words, both genetic and environmental correlations between the ASPD criteria could be accounted for by a single common latent variable. The findings support the validity of ASPD as a unidimensional diagnostic construct.
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