Evidence map›Paper›PMID 28105583›Full record

ArticleThe journal of physiological sciences : JPS2018

TRPC3 participates in angiotensin II type 1 receptor-dependent stress-induced slow increase in intracellular Ca

Yohei Yamaguchi, Gentaro Iribe, Toshiyuki Kaneko, Ken Takahashi, Takuro Numaga-Tomita, Motohiro Nishida, Lutz Birnbaumer, Keiji Naruse

Open access · goldAbstract read
In one paragraph

Article in The journal of physiological sciences : JPS, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
2.3field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 31 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Myocardial TRPC6-mediated ZnNature communications · 2022
    Article
  5. Review
  6. Article
  7. Effects of Sarcolemmal Background CaFrontiers in physiology · 2022
    Article
  8. Review
  9. Article
  10. Review
  11. Article
  12. Review
  13. Article
  14. Article
  15. Article
  16. Review
  17. Article
  18. Impact of titin strain on the cardiac slow force response.Progress in biophysics and molecular biology · 2017
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 5 institutions in 2 countries.

Yohei YamaguchiDepartment of Cardiovascular Physiology, Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama University, Okayama, 700-8558, Japan.
Gentaro IribeDepartment of Cardiovascular Physiology, Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama University, Okayama, 700-8558, Japan. iribe@okayama-u.ac.jp.
Toshiyuki KanekoDepartment of Physiology, Asahikawa Medical University, Asahikawa, Hokkaido, 078-8510, Japan.
Ken TakahashiDepartment of Cardiovascular Physiology, Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama University, Okayama, 700-8558, Japan.
Takuro Numaga-TomitaDivision of Cardiocirculatory Signaling, Okazaki Institute for Integrative Bioscience (National Institute for Physiological Sciences), National Institutes of Natural Sciences, Okazaki, Aichi, 444-8787, Japan.
Motohiro NishidaDivision of Cardiocirculatory Signaling, Okazaki Institute for Integrative Bioscience (National Institute for Physiological Sciences), National Institutes of Natural Sciences, Okazaki, Aichi, 444-8787, Japan.
Lutz BirnbaumerNeurobiology Laboratory, National Institute of Environmental Health Science, Research Triangle Park, NC, 27709, USA.
Keiji NaruseDepartment of Cardiovascular Physiology, Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama University, Okayama, 700-8558, Japan.
Okayama University · JPAsahikawa Medical University · JPNational Institute for Physiological Sciences · JPNational Institutes of Natural Sciences · JPTriangle · US

Funding

Intramural Research Program of the NIH Z01-ES-101864Japan Society for the Promotion of Science 23300167, 26282121
6 · The paper itself

Abstract

When a cardiac muscle is held in a stretched position, its [Ca

Indexed as

Angiotensin IIAnimalsCalciumMiceMice, Inbred C57BLMice, KnockoutMyocardiumMyocytes, CardiacReceptor, Angiotensin, Type 1TRPC Cation ChannelsAngiotensin IICalciumReceptor, Angiotensin, Type 1TRPC3 cation channelTRPC Cation ChannelsAngiotensin II type 1 receptorCa2+ handlingCardiomyocyteMathematical modelStretchTransient receptor potential canonical 3

Identifiers

PMID28105583
PMCPMC10718017
OpenAlexW2578741777

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.