Evidence map›Paper›PMID 28102054›Full record

ReviewAllergy, asthma & immunology research2017

Extracellular Vesicles, a Key Mediator to Link Environmental Microbiota to Airway Immunity.

Youngwoo Choi, Hanki Park, Hae Sim Park, Yoon Keun Kim

Abstract readReview
In one paragraph

Review in Allergy, asthma & immunology research, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed.

  1. Review
  2. Article
  3. New targets for type 2-low asthma.The Korean journal of internal medicine · 2024
    Review
  4. Article
  5. Review
  6. Article
  7. Article
  8. Article
  9. Review
  10. Review
  11. Article
  12. Review
  13. Article
  14. Pulmonary Surfactants: a New Therapeutic Target in Asthma.Current allergy and asthma reports · 2020
    Review
  15. Article
  16. Review
  17. Nasal Microbiota Modifies the Effects of Particulate Air Pollution on Plasma Extracellular Vesicles.International journal of environmental research and public health · 2020
    Article
  18. Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Youngwoo ChoiDepartment of Allergy and Clinical Immunology, Ajou University Medical Center, Suwon, Korea.
Hanki ParkDepartment of Allergy and Clinical Immunology, Ajou University Medical Center, Suwon, Korea.
Hae Sim ParkDepartment of Allergy and Clinical Immunology, Ajou University Medical Center, Suwon, Korea. hspark@ajou.ac.kr.ORCID http://orcid.org/0000-0003-2614-0303
Yoon Keun KimInstitute of MD Healthcare, Seoul, Korea. juinea2215@naver.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Asthma is considered the hallmark of chronic airway inflammation, in which several inflammatory cells of the innate and adaptive immune system act together. The disease is thought to be caused by a combination of genetic and environmental factors; however, precise mechanisms for airway inflammation remain unclear. The human microbiota provides an increasingly favored explanation for inflammatory diseases; an altered microbiota composition has been shown to regulate immune responses. However, given the complexity of the microbiota, additional research is needed to elucidate its role in the development of disease. One of the candidate molecules that link microbiota to disease is the extracellular vesicles (EVs). EVs are secreted by diverse cell types and they possess the pathophysiological function of delivering signals between bacteria and host. We discuss the role of the microbiota in the development of asthma through releasing EVs.

Indexed as

Asthmaextracellular vesiclesinflammationmicrobiota

Identifiers

PMID28102054
PMCPMC5266118

What OpenQuestion holds

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LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.