Evidence map›Paper›PMID 28075048›Full record

ArticleJournal of cellular biochemistry2017

The STI and UBA Domains of UBQLN1 Are Critical Determinants of Substrate Interaction and Proteostasis.

Zimple Kurlawala, Parag P Shah, Charmi Shah, Levi J Beverly

Open access · greenAbstract read
In one paragraph

Article in Journal of cellular biochemistry, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 41 papers.

0numbers the graph read from it
0cells of the map it votes in
41citing papers in PubMed
1.9field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

41 citing papers in PubMed, 56 citations in OpenAlex.

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  17. TheActa biochimica et biophysica Sinica · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Zimple KurlawalaJames Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky.
Parag P ShahJames Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky.
Charmi ShahJames Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky.
Levi J BeverlyJames Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky.
James Graham Brown Foundation · USUniversity of Louisville · US

Funding

Ubiquilins regulate EMT, migration and invasion of human lung cancerR01CA193220 · NCI · UNIVERSITY OF LOUISVILLE · PI BEVERLY, LEVI J · 2015 to 2019
$2.3M
Molecular Targets COBREP20GM103482 · NIGMS · UNIVERSITY OF LOUISVILLE · PI MILLER, DONALD M. · 2012 to 2012
$2.0M
NCI NIH HHS R01 CA193220NIGMS NIH HHS P20 GM103482
6 · The paper itself

Abstract

There are five Ubiquilin proteins (UBQLN1-4, UBQLN-L), which are evolutionarily conserved and structurally similar. UBQLN proteins have three functional domains: N-terminal ubiquitin-like domain (UBL), C-terminal ubiquitin-associated domain (UBA), and STI chaperone-like regions in the middle. Alterations in UBQLN1 gene have been detected in a variety of disorders ranging from Alzheimer's disease to cancer. UBQLN1 has been largely studied in neurodegenerative disorders in the context of protein quality control. Several studies have hypothesized that the UBA domain of UBQLN1 binds to poly-ubiquitin chains of substrate and shuttles it to the proteasome via its UBL domain for degradation. UBQLN1 either facilitates degradation (Ataxin3, EPS15) or stabilizes (PSEN1/2, BCLb) substrates it binds to. The signal that determines this fate is unknown and there is conflicting data to support the existing working model of UBQLN1. Using BCLb as a model substrate, we characterized UBQLN1-substrate interaction. We identified the first two STI domains of UBQLN1 as critical for binding to BCLb. Interaction of UBQLN1 with BCLb is independent of ubiquitination of BCLb, but interaction with ubiquitin via UBA domain is required for stabilization of BCLb. Similarly, we showed that UBQLN1 interacts with IGF1R and ESYT2 through the STI domains and stabilizes these proteins through its UBA domain. Interactions that are not dependent on STI domains, for example, UBL mediated interaction with PSMD4 and BAG6, do not appear to be stabilized by UBQLN1. We conclude that fate of substrates that UBQLN1 associates with, is interaction domain specific. J. Cell. Biochem. 118: 2261-2270, 2017. © 2017 Wiley Periodicals, Inc.

Indexed as

Adaptor Proteins, Signal TransducingAutophagy-Related ProteinsCarrier ProteinsCell Cycle ProteinsCell LineCytoplasmHumansMass SpectrometryMolecular ChaperonesPlasmidsProteasome Endopeptidase ComplexProtein BindingProtein Structure, TertiaryProteostasisProto-Oncogene Proteins c-bcl-2Receptor, IGF Type 1Adaptor Proteins, Signal TransducingAutophagy-Related ProteinsBAG6 protein, humanBCL2L14 protein, humanCarrier ProteinsCell Cycle ProteinsIGF1R protein, humanMolecular ChaperonesProteasome Endopeptidase ComplexProto-Oncogene Proteins c-bcl-2PSMD4 protein, humanReceptor, IGF Type 1Receptors, SomatomedinRNA-Binding ProteinsUbiquitinUBQLN1 protein, humanBAG6BCLbESYT2IGF1RPROTEOSTASISPSMD4STIUBAUBLUBQLN

Identifiers

PMID28075048
PMCPMC5462870
OpenAlexW2579676443

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.